Evidence map›Paper›PMID 28039475›Full record

ArticleOncotarget2017

GPR120, a potential therapeutic target for experimental colitis in IL-10 deficient mice.

Jie Zhao, Honggang Wang, Peiliang Shi, Wenbo Wang, Ye Sun

Open access · diamondAbstract read
In one paragraph

Article in Oncotarget, 2017. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 9 papers.

0numbers the graph read from it
0cells of the map it votes in
9citing papers in PubMed
1.2field-weighted citation impact, top 20% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

9 citing papers in PubMed, 19 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 4 institutions in 1 country.

Jie ZhaoDepartment of General Surgery, The First Affiliated Hospital of Soochow University, Suzhou, 215000, Jiangsu, China.
Honggang WangDepartment of General Surgery, Taizhou People's Hospital, Medical School of Nantong University, Taizhou, 225300, Jiangsu, China.
Peiliang ShiMOE Key Laboratory of Model Animal for Disease Study, Model Animal Research Center, Nanjing University, Nanjing, 210000, Jiangsu, China.
Wenbo WangDepartment of General Surgery, The First Affiliated Hospital of Soochow University, Suzhou, 215000, Jiangsu, China.
Ye SunDepartment of Orthopedics, The First Affiliated Hospital of Soochow University, Suzhou, 215000, Jiangsu, China.
Soochow University · CNFirst Affiliated Hospital of Soochow University · CNNanjing University · CNTaizhou People's Hospital · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

It has been proved that interleukin-10-knockout (IL-10 KO) mice display the most similar characteristics to that of human Crohn's disease (CD). Docosahexaenoic acid (DHA) has well established beneficial effects on human and animal models health with potent anti-inflammatory effects with poorly understood mechanisms. This study was aimed at figuring out whether DHA could ameliorate the Crohn's colitis by activating GPR120 and whether GPR120 could be a potential therapeutic target for CD.16 week-old mice included in our present study were divided into three groups, WT group, IL-10 KO group and DHA group(IL-10 KO mice with DHA treatment, i.g., 35.5mg/kg/d), containing 8 mice in each group. The severity of colitis, pro-inflammatory cytokines concentrations, the expression/distribution of protein GPR120 and TAK1/IKK-α/IkB-α/p65 pathway in the proximal colons were evaluated at the end of the experiment. Administration of DHA showed promising results in the experimental chronic colitis (demonstrated by reduced infiltration of inflammatory cells, lowered inflammation scores, decreased pro-inflammatory cytokines) and body weight loss improvement. Moreover, in the DHA-treated mice, enhanced expression and improved distribution integrity of protein GPR120 were observed, which was probably associated with the regulation of TAK1/IKK-α/IkB-α/p65 pathway. Our results indicated that triggering GPR120 via the inhibition of TAK1/IKK-α/IkB-α/p65 pathway might be an important target for Crohn's colitis.

Indexed as

AnimalsAnti-Inflammatory AgentsColitisColonCytokinesDisease Models, AnimalDocosahexaenoic AcidsGastrointestinal AgentsGenetic Predisposition to DiseaseInflammation MediatorsInterleukin-10Mice, Inbred C57BLMice, KnockoutMolecular Targeted TherapyPhenotypeReceptors, G-Protein-CoupledAnti-Inflammatory AgentsCytokinesDocosahexaenoic AcidsFFAR4 protein, mouseGastrointestinal AgentsIL10 protein, mouseInflammation MediatorsInterleukin-10Receptors, G-Protein-CoupledcolitisCrohn’s diseasedocosahexaenoic acidGPR120TAK1/IKK-α/IkB-α/p65 pathway

Identifiers

PMID28039475
PMCPMC5352409
OpenAlexW2564732232

What OpenQuestion holds

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LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.