Evidence map›Paper›PMID 28007846›Full record

ArticleJournal of lipid research2017

Different origins of lysophospholipid mediators between coronary and peripheral arteries in acute coronary syndrome.

Makoto Kurano, Kuniyuki Kano, Tomotaka Dohi, Hirotaka Matsumoto, Koji Igarashi, Masako Nishikawa, Ryunosuke Ohkawa, Hitoshi Ikeda, Katsumi Miyauchi, Hiroyuki Daida and 2 more

Open access · hybridAbstract read
In one paragraph

Article in Journal of lipid research, 2017. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 15 papers.

0numbers the graph read from it
0cells of the map it votes in
15citing papers in PubMed
2.6field-weighted citation impact, top 10% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

15 citing papers in PubMed, 38 citations in OpenAlex.

  1. Article
  2. Article
  3. Article
  4. Lipoprotein(a): structural basis, bidirectional risk, and therapeutic frontiers.Journal of clinical biochemistry and nutrition · 2026
    Article
  5. Article
  6. Article
  7. LPAJournal of inflammation research · 2023
    Article
  8. Article
  9. Article
  10. Article
  11. Article
  12. Article
  13. Article
  14. Lysophospholipids in laboratory medicine.Proceedings of the Japan Academy. Series B, Physical and biological sciences · 2018
    Review
  15. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

12 authors at 6 institutions in 1 country.

Makoto KuranoDepartment of Clinical Laboratory Medicine, Graduate School of Medicine, University of Tokyo, Tokyo, Japan.
Kuniyuki KanoCREST, Japan Science and Technology Corporation (JST).
Tomotaka DohiDepartment of Cardiovascular Medicine, Juntendo University School of Medicine, Tokyo, Japan.
Hirotaka MatsumotoLaboratory of Molecular and Cellular Biochemistry, Graduate School of Pharmaceutical Sciences, Tohoku University, Miyagi, Japan.
Koji IgarashiBioscience Division, Reagent Development Department, AIA Research Group, TOSOH Corporation, Kanagawa, Japan.
Masako NishikawaDepartment of Clinical Laboratory Medicine, Graduate School of Medicine, University of Tokyo, Tokyo, Japan.
Ryunosuke OhkawaDepartment of Clinical Laboratory, University of Tokyo Hospital, Tokyo, Japan.
Hitoshi IkedaDepartment of Clinical Laboratory Medicine, Graduate School of Medicine, University of Tokyo, Tokyo, Japan.
Katsumi MiyauchiDepartment of Cardiovascular Medicine, Juntendo University School of Medicine, Tokyo, Japan.
Hiroyuki DaidaDepartment of Cardiovascular Medicine, Juntendo University School of Medicine, Tokyo, Japan.
Junken AokiCREST, Japan Science and Technology Corporation (JST).
Yutaka YatomiDepartment of Clinical Laboratory Medicine, Graduate School of Medicine, University of Tokyo, Tokyo, Japan yatoyuta-tky@umin.ac.jp.
Japan Science and Technology Agency · JPJuntendo University · JPThe University of Tokyo · JPTohoku University · JPTosoh (Japan) · JPUniversity of Tokyo Hospital · JP

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Lysophosphatidic acids (LysoPAs) and lysophosphatidylserine (LysoPS) are emerging lipid mediators proposed to be involved in the pathogenesis of acute coronary syndrome (ACS). In this study, we attempted to elucidate how LysoPA and LysoPS become elevated in ACS using human blood samples collected simultaneously from culprit coronary arteries and peripheral arteries in ACS subjects. We found that: 1) the plasma LysoPA, LysoPS, and lysophosphatidylglycerol levels were not different, while the lysophosphatidylcholine (LysoPC), lysophosphatidylinositol, and lysophosphatidylethanolamine (LysoPE) levels were significantly lower in the culprit coronary arteries; 2) the serum autotaxin (ATX) level was lower and the serum phosphatidylserine-specific phospholipase A

Indexed as

Acute Coronary SyndromeAtherosclerosisCoronary VesselsFemaleHeartHumansLysophospholipase DLysophospholipidsMaleMass SpectrometryPhospholipases A1Phosphoric Diester HydrolaseslysophosphatidylglycerollysophosphatidylinositollysophosphatidylserineLysophospholipase DLysophospholipidsPhospholipases A1Phosphoric Diester Hydrolasesacute coronary diseaselysophosphatidic acidslysophosphatidylcholinelysophosphatidylethanolaminelysophosphatidylserine

Identifiers

PMID28007846
PMCPMC5282959
OpenAlexW2561473054

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.