Evidence map›Paper›PMID 27998954›Full record

ArticleEndocrine connections2017

Glucagon-producing cells are increased in Mas-deficient mice.

Janaína Felix Braga, Daniela Ravizzoni Dartora, Natalia Alenina, Michael Bader, Robson Augusto Souza Santos

Abstract read
PubMed Full text at PMC Publisher
In one paragraph

Article in Endocrine connections, 2017. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed.

  1. Article
  2. The antiobese effect of AT1 receptor blockade is augmented in mice lacking Mas.Naunyn-Schmiedeberg's archives of pharmacology · 2019
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors.

Janaína Felix BragaDepartment of Physiology and BiophysicsNational Institute of Science and Technology in Nanobiopharmaceutics, Institute of Biological Sciences, Federal University of Minas Gerais, Belo Horizonte, Minas Gerais, Brazil.
Daniela Ravizzoni DartoraCardiology Institute of Rio Grande do Sul/University Foundation of Cardiology (IC/FUC)Porto Alegre, Rio Grande do Sul, Brazil.
Natalia AleninaMax-Delbruck Center of Molecular Medicine (MDC)Berlin-Buch, Berlin, Germany.
Michael BaderCardiology Institute of Rio Grande do Sul/University Foundation of Cardiology (IC/FUC)Porto Alegre, Rio Grande do Sul, Brazil.
Robson Augusto Souza SantosDepartment of Physiology and BiophysicsNational Institute of Science and Technology in Nanobiopharmaceutics, Institute of Biological Sciences, Federal University of Minas Gerais, Belo Horizonte, Minas Gerais, Brazil robsonsant@gmail.com.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

It has been shown that angiotensin(1-7) (Ang(1-7)) produces several effects related to glucose homeostasis. In this study, we aimed to investigate the effects of genetic deletion of Ang(1-7), the GPCR Mas, on the glucagon-producing cells. C57BL6/N Mas

Indexed as

diabetesmetabolism

Identifiers

PMID27998954
PMCPMC5302165

What OpenQuestion holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.