ArticleEvolution; international journal of organic evolution2017
Sexual selection modulates genetic conflicts and patterns of genomic imprinting.
Article in Evolution; international journal of organic evolution, 2017. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers.
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Who cites it
5 citing papers in PubMed.
- No evidence that relatedness or familiarity modulates male harm inEcology and evolution · 2022Article
- Sex-biased demography modulates male harm across the genome.Proceedings. Biological sciences · 2021Article
- The social evolution of sleep: sex differences, intragenomic conflicts and clinical pathologies.Proceedings. Biological sciences · 2019Article
- Why war is a man's game.Proceedings. Biological sciences · 2018Article
- Male relatedness and familiarity are required to modulate male-induced harm to females inProceedings. Biological sciences · 2017Article
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3 authors.
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Abstract
Recent years have seen a surge of interest in linking the theories of kin selection and sexual selection. In particular, there is a growing appreciation that kin selection, arising through demographic factors such as sex-biased dispersal, may modulate sexual conflicts, including in the context of male-female arms races characterized by coevolutionary cycles. However, evolutionary conflicts of interest need not only occur between individuals, but may also occur within individuals, and sex-specific demography is known to foment such intragenomic conflict in relation to social behavior. Whether and how this logic holds in the context of sexual conflict-and, in particular, in relation to coevolutionary cycles-remains obscure. We develop a kin-selection model to investigate the interests of different genes involved in sexual and intragenomic conflict, and we show that consideration of these conflicting interests yields novel predictions concerning parent-of-origin specific patterns of gene expression and the detrimental effects of different classes of mutation and epimutation at loci underpinning sexually selected phenotypes.
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