Evidence map›Paper›PMID 27986928›Full record

ArticleThe Journal of neuroscience : the official journal of the Society for Neuroscience2017

Presynaptic Neuronal Pentraxin Receptor Organizes Excitatory and Inhibitory Synapses.

Sung-Jin Lee, Mengping Wei, Chen Zhang, Stephan Maxeiner, ChangHui Pak, Salome Calado Botelho, Justin Trotter, Fredrik H Sterky, Thomas C Südhof

Open access · bronzeAbstract read
In one paragraph

Article in The Journal of neuroscience : the official journal of the Society for Neuroscience, 2017. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 100 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
100citing papers in PubMed, 1 pooled it
4.0field-weighted citation impact, top 6% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

100 citing papers in PubMed, 1 synthesis or guideline pooled it, 160 citations in OpenAlex.

  1. Pooled it
  2. Declining levels of functionally specialized synaptic proteins in plasma neuronal exosomes with progression of Alzheimer's disease.FASEB journal : official publication of the Federation of American Societies for Experimental Biology · 2018
    Trial
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  8. The neuroimmune system and cognition.Nature immunology · 2026
    Review
  9. Article
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  11. Dynamic extracellular interactions with AMPA receptors.Proceedings of the National Academy of Sciences of the United States of America · 2025
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  13. Review
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40 more citing papers are in PubMed but not listed here.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors at 3 institutions in 2 countries.

Sung-Jin LeeDepartment of Molecular and Cellular Physiology, Stanford University School of Medicine, Stanford, California 94305, sungjinl@stanford.edu tcs1@stanford.edu.
Mengping WeiState Key Laboratory of Biomembrane and Membrane Biotechnology, School of Life Sciences, Peking University, Beijing 100871, China, and.
Chen ZhangState Key Laboratory of Biomembrane and Membrane Biotechnology, School of Life Sciences, Peking University, Beijing 100871, China, and.
Stephan MaxeinerDepartment of Molecular and Cellular Physiology, Stanford University School of Medicine, Stanford, California 94305.ORCID 0000-0002-4472-695X
ChangHui PakDepartment of Molecular and Cellular Physiology, Stanford University School of Medicine, Stanford, California 94305.
Salome Calado BotelhoDepartment of Molecular and Cellular Physiology, Stanford University School of Medicine, Stanford, California 94305.ORCID 0000-0003-2822-2257
Justin TrotterDepartment of Molecular and Cellular Physiology, Stanford University School of Medicine, Stanford, California 94305.
Fredrik H SterkyDepartment of Molecular and Cellular Physiology, Stanford University School of Medicine, Stanford, California 94305.ORCID 0000-0001-8881-0523
Thomas C SüdhofDepartment of Molecular and Cellular Physiology, Stanford University School of Medicine, Stanford, California 94305, sungjinl@stanford.edu tcs1@stanford.edu.
Stanford University · USPeking University · CNHoward Hughes Medical Institute · US

Funding

FUNCTION OF NEUREXINSR37MH052804 · NIMH · UT SOUTHWESTERN MEDICAL CENTER · PI SUDHOF, THOMAS C. · 2000 to 2019
$9.3M
Function of NeurexinsR01MH052804 · NIMH · UNIVERSITY OF TEXAS SW MED CTR/DALLAS · PI SUDHOF, THOMAS C. · 1994 to 2024
$3.7M
Investigating role of neurexin-1 mutation in autism using human induced neuronsF32HD078051 · NICHD · STANFORD UNIVERSITY · PI PAK, CHANGHUI · 2013 to 2015
$159k
NICHD NIH HHS F32 HD078051NIMH NIH HHS R01 MH052804NIMH NIH HHS R37 MH052804
6 · The paper itself

Abstract

Three neuronal pentraxins are expressed in brain, the membrane-bound "neuronal pentraxin receptor" (NPR) and the secreted proteins NP1 and NARP (i.e., NP2). Neuronal pentraxins bind to AMPARs at excitatory synapses and play important, well-documented roles in the activity-dependent regulation of neural circuits via this binding activity. However, it is unknown whether neuronal pentraxins perform roles in synapses beyond modulating postsynaptic AMPAR-dependent plasticity, and whether they may even act in inhibitory synapses. Here, we show that NPR expressed in non-neuronal cells potently induces formation of both excitatory and inhibitory postsynaptic specializations in cocultured hippocampal neurons. Knockdown of NPR in hippocampal neurons, conversely, dramatically decreased assembly and function of both excitatory and inhibitory postsynaptic specializations. Overexpression of NPR rescued the NPR knockdown phenotype but did not in itself change synapse numbers or properties. However, the NPR knockdown decreased the levels of NARP, whereas NPR overexpression produced a dramatic increase in the levels of NP1 and NARP, suggesting that NPR recruits and stabilizes NP1 and NARP on the presynaptic plasma membrane. Mechanistically, NPR acted in excitatory synapse assembly by binding to the N-terminal domain of AMPARs; antagonists of AMPA and GABA receptors selectively inhibited NPR-induced heterologous excitatory and inhibitory synapse assembly, respectively, but did not affect neurexin-1β-induced synapse assembly as a control. Our data suggest that neuronal pentraxins act as signaling complexes that function as general trans-synaptic organizers of both excitatory and inhibitory synapses by a mechanism that depends, at least in part, on the activity of the neurotransmitter receptors at these synapses. SIGNIFICANCE STATEMENT: Neuronal pentraxins comprise three neuronal proteins, neuronal pentraxin receptor (NPR) which is a type-II transmembrane protein on the neuronal surface, and secreted neuronal pentraxin-1 and NARP. The general functions of neuronal pentraxins at synapses have not been explored, except for their basic AMPAR binding properties. Here, we examined the functional role of NPR at synapses because it is the only neuronal pentraxin that is anchored to the neuronal cell-surface membrane. We find that NPR is a potent inducer of both excitatory and inhibitory heterologous synapses, and that knockdown of NPR in cultured neurons decreases the density of both excitatory and inhibitory synapses. Our data suggest that NPR performs a general, previously unrecognized function as a universal organizer of synapses.

Indexed as

AnimalsCoculture TechniquesC-Reactive ProteinExcitatory Amino Acid AntagonistsExcitatory Postsynaptic PotentialsGABA AntagonistsGene Knockdown TechniquesHEK293 CellsHippocampusHumansMiceNerve Tissue ProteinsNeuronsPatch-Clamp TechniquesPentraxinsReceptors, AMPAC-Reactive ProteinExcitatory Amino Acid AntagonistsGABA AntagonistsNerve Tissue ProteinsPentraxinsReceptors, AMPAReceptors, Cell SurfaceRNA, Small InterferingAMPARcell adhesion moleculeneuronal pentraxin receptorsynaptogenesis

Identifiers

PMID27986928
PMCPMC5296791
OpenAlexW2562584466

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.