Evidence map›Paper›PMID 27981013›Full record

ArticleAdvanced science (Weinheim, Baden-Wurttemberg, Germany)2016

PLGA-PNIPAM Microspheres Loaded with the Gastrointestinal Nutrient NaB Ameliorate Cardiac Dysfunction by Activating Sirt3 in Acute Myocardial Infarction.

Panke Cheng, Wen Zeng, Li Li, Da Huo, Lingqing Zeng, Ju Tan, Jingting Zhou, Jiansen Sun, Ge Liu, Yanzhao Li and 3 more

Open access · goldAbstract read
In one paragraph

Article in Advanced science (Weinheim, Baden-Wurttemberg, Germany), 2016. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 14 papers.

0numbers the graph read from it
0cells of the map it votes in
14citing papers in PubMed
1.9field-weighted citation impact, top 15% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

14 citing papers in PubMed, 30 citations in OpenAlex.

  1. Review
  2. Review
  3. Review
  4. Article
  5. Review
  6. Review
  7. Article
  8. Article
  9. SIRT3: A New Regulator of Cardiovascular Diseases.Oxidative medicine and cellular longevity · 2018
    Review
  10. Article
  11. Article
  12. Knockdown of MicroRNA-122 Protects H9c2 Cardiomyocytes from Hypoxia-Induced Apoptosis and Promotes Autophagy.Medical science monitor : international medical journal of experimental and clinical research · 2017
    Article
  13. Article
  14. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

13 authors at 1 institution in 1 country.

Panke ChengDepartment of Anatomy National & Regional Engineering Laboratory of Tissue Engineering State and Local Joint Engineering Laboratory For Vascular Implants, Key Lab for Biomechanics and Tissue Engineering of Chongqing Third Military Medical University Chongqing 400038 China.
Wen ZengDepartment of Anatomy National & Regional Engineering Laboratory of Tissue Engineering State and Local Joint Engineering Laboratory For Vascular Implants, Key Lab for Biomechanics and Tissue Engineering of Chongqing Third Military Medical University Chongqing 400038 China.
Li LiDepartment of Anatomy National & Regional Engineering Laboratory of Tissue Engineering State and Local Joint Engineering Laboratory For Vascular Implants, Key Lab for Biomechanics and Tissue Engineering of Chongqing Third Military Medical University Chongqing 400038 China.
Da HuoDepartment of Anatomy National & Regional Engineering Laboratory of Tissue Engineering State and Local Joint Engineering Laboratory For Vascular Implants, Key Lab for Biomechanics and Tissue Engineering of Chongqing Third Military Medical University Chongqing 400038 China.
Lingqing ZengDepartment of Anatomy National & Regional Engineering Laboratory of Tissue Engineering State and Local Joint Engineering Laboratory For Vascular Implants, Key Lab for Biomechanics and Tissue Engineering of Chongqing Third Military Medical University Chongqing 400038 China.
Ju TanDepartment of Anatomy National & Regional Engineering Laboratory of Tissue Engineering State and Local Joint Engineering Laboratory For Vascular Implants, Key Lab for Biomechanics and Tissue Engineering of Chongqing Third Military Medical University Chongqing 400038 China.
Jingting ZhouDepartment of Anatomy National & Regional Engineering Laboratory of Tissue Engineering State and Local Joint Engineering Laboratory For Vascular Implants, Key Lab for Biomechanics and Tissue Engineering of Chongqing Third Military Medical University Chongqing 400038 China.
Jiansen SunDepartment of Anatomy National & Regional Engineering Laboratory of Tissue Engineering State and Local Joint Engineering Laboratory For Vascular Implants, Key Lab for Biomechanics and Tissue Engineering of Chongqing Third Military Medical University Chongqing 400038 China.
Ge LiuDepartment of Anatomy National & Regional Engineering Laboratory of Tissue Engineering State and Local Joint Engineering Laboratory For Vascular Implants, Key Lab for Biomechanics and Tissue Engineering of Chongqing Third Military Medical University Chongqing 400038 China.
Yanzhao LiDepartment of Anatomy National & Regional Engineering Laboratory of Tissue Engineering State and Local Joint Engineering Laboratory For Vascular Implants, Key Lab for Biomechanics and Tissue Engineering of Chongqing Third Military Medical University Chongqing 400038 China.
Ge GuanDepartment of Anatomy National & Regional Engineering Laboratory of Tissue Engineering State and Local Joint Engineering Laboratory For Vascular Implants, Key Lab for Biomechanics and Tissue Engineering of Chongqing Third Military Medical University Chongqing 400038 China.
Yuxin WangDepartment of Anatomy National & Regional Engineering Laboratory of Tissue Engineering State and Local Joint Engineering Laboratory For Vascular Implants, Key Lab for Biomechanics and Tissue Engineering of Chongqing Third Military Medical University Chongqing 400038 China.
Chuhong ZhuDepartment of Anatomy National & Regional Engineering Laboratory of Tissue Engineering State and Local Joint Engineering Laboratory For Vascular Implants, Key Lab for Biomechanics and Tissue Engineering of Chongqing Third Military Medical University Chongqing 400038 China.
Army Medical University · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Acute myocardial infarction (AMI) is the death of cardiomyocytes caused by a lack of energy due to ischemia. Nutrients supplied by the blood are the main source of cellular energy for cardiomyocytes. Sodium butyrate (NaB), a gastrointestinal nutrient, is a short-chain fatty acid (butyric acid) that may act as an energy source in AMI therapy. Poly(lactic-co-glycolic acid)-Poly (

Indexed as

fatty acidsischemiamicrospheresSirt3

Identifiers

PMID27981013
PMCPMC5157182
OpenAlexW2538805822

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.