Evidence map›Paper›PMID 27974128›Full record

ReviewZhongguo dang dai er ke za zhi = Chinese journal of contemporary pediatrics2016

[Research advances in susceptible genes for developmental dyslexia in children].

Rui Kong, Ran-Ran Song

Abstract readReview
PubMed Full text at PMC
In one paragraph

Review in Zhongguo dang dai er ke za zhi = Chinese journal of contemporary pediatrics, 2016. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors.

Rui KongDepartment of Maternal and Child Health Care, School of Public Health, Tongji Medical College, Huazhong University of Science and Technology, Wuhan 430030, China. songranran@hust.edu.cn.
Ran-Ran Song

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Developmental dyslexia in children is one of the neurodevelopmental disorders and is affected by various susceptible genes. In recent years, researchers have found some susceptible genes for dyslexia via chromosome analysis, genome-wide association studies, association analysis, gene function research, neuroimaging, and neurophysiological techniques. This article reviews the research advances in susceptible genes for developmental dyslexia, and with the study on susceptible genes for dyslexia, it lays a foundation for in-depth studies on the "gene-brain-behavior" level and provides scientific clues for exploring etiology and pathogenesis of dyslexia.

Indexed as

Genetic Predisposition to DiseaseChildCytoskeletal ProteinsDyslexiaForkhead Transcription FactorsHumansMicrotubule-Associated ProteinsNerve Tissue ProteinsNuclear ProteinsReceptors, ImmunologicRoundabout ProteinsCytoskeletal ProteinsDCDC2 protein, humanDNAAF4 protein, humanForkhead Transcription FactorsFOXP2 protein, humanKIAA0319 protein, humanMicrotubule-Associated ProteinsNerve Tissue ProteinsNuclear ProteinsReceptors, ImmunologicRoundabout Proteins

Identifiers

PMID27974128
PMCPMC7403085

What OpenQuestion holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.