Evidence map›Paper›PMID 27867530›Full record

ArticleESC heart failure2016

Continuous renal replacement therapy for haemodynamic collapse and rhabdomyolysis induced by pheochromocytoma crisis.

Xi Zhou, Chuhuan Zhao, Xiafei Feng, Anweshan Samanta, Yi Nuo Lin, Jun Chen, Xiaochun Dai, Xia Hong, Qiangli Xie, Weijian Huang

Open access · goldAbstract read
In one paragraph

Article in ESC heart failure, 2016. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.

0numbers the graph read from it
0cells of the map it votes in
3citing papers in PubMed
0.3field-weighted citation impact, top 33% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

3 citing papers in PubMed, 4 citations in OpenAlex.

  1. Article
  2. Article
  3. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

10 authors at 3 institutions in 2 countries.

Xi ZhouDepartment of Cardiac Care UnitThe First Affiliated Hospital of Wenzhou Medical UniversityWenzhouChina; Department of Cardiology, The Key Lab of Cardiovascular Disease of WenzhouThe First Affiliated Hospital of Wenzhou Medical UniversityWenzhouChina.
Chuhuan ZhaoDepartment of Cardiac Care Unit The First Affiliated Hospital of Wenzhou Medical University Wenzhou China.
Xiafei FengDepartment of Cardiac Care UnitThe First Affiliated Hospital of Wenzhou Medical UniversityWenzhouChina; Department of Cardiology, The Key Lab of Cardiovascular Disease of WenzhouThe First Affiliated Hospital of Wenzhou Medical UniversityWenzhouChina.
Anweshan SamantaInstitute of Cardiovascular Research University of Kansas Medical Center Kansas City KS USA.
Yi Nuo LinDepartment of Cardiology, The Key Lab of Cardiovascular Disease of Wenzhou The First Affiliated Hospital of Wenzhou Medical University Wenzhou China.
Jun ChenDepartment of Cardiac Care Unit The First Affiliated Hospital of Wenzhou Medical University Wenzhou China.
Xiaochun DaiDepartment of Cardiac Care Unit The First Affiliated Hospital of Wenzhou Medical University Wenzhou China.
Xia HongDepartment of Cardiac Care Unit The First Affiliated Hospital of Wenzhou Medical University Wenzhou China.
Qiangli XieDepartment of Cardiac Care Unit The First Affiliated Hospital of Wenzhou Medical University Wenzhou China.
Weijian HuangDepartment of Cardiac Care UnitThe First Affiliated Hospital of Wenzhou Medical UniversityWenzhouChina; Department of Cardiology, The Key Lab of Cardiovascular Disease of WenzhouThe First Affiliated Hospital of Wenzhou Medical UniversityWenzhouChina.
First Affiliated Hospital of Wenzhou Medical University · CNWenzhou Medical University · CNUniversity of Kansas Medical Center · US

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Pheochromocytoma associated with pregnancy is not common. Caesarean section may induce pheochromocytoma crisis, resulting in a lethal condition. The clinical picture of pheochromocytoma crisis is extremely variable. In this report, we describe a case of severe pheochromocytoma crisis induced by caesarean section presenting with hyperpyrexia, haemodynamic collapse, muscle weakness, heart failure, and acute kidney injury. Furthermore, we report that the muscle weakness was a manifestation of rhabdomyolysis, resulting from the pheochromocytoma crisis. Standard medical therapy failed to halt the patient's rapidly deteriorating condition. Continuous renal replacement therapy removed catecholamines from the circulation, resulting in improvement of haemodynamics and abrogation of rhabdomyolysis.

Indexed as

Continuous renal replacement therapyHaemodynamic collapsePheochromocytoma crisisRhabdomyolysis

Identifiers

PMID27867530
PMCPMC5107978
OpenAlexW2514043352

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.