ArticleMedicine2016
Delayed gait recovery with recovery of an injured corticoreticulospinal tract in a chronic hemiparetic patient: A case report.
Article in Medicine, 2016. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 6 papers.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
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Who cites it
6 citing papers in PubMed.
- Alpha and beta/low-gamma frequency bands may have distinct neural origin and function during post-stroke walking.Experimental brain research · 2024Article
- The Relationship between Structure of the Corticoreticular Tract and Walking Capacity in Children with Cerebral Palsy.Journal of biomedical physics & engineering · 2024Article
- Balance Rehabilitation Approach by Bobath and Vojta Methods in Cerebral Palsy: A Pilot Study.Children (Basel, Switzerland) · 2022Article
- Is there frequency-specificity in the motor control of walking? The putative differential role of alpha and beta oscillations.Frontiers in systems neuroscience · 2022Review
- Corticoreticular Pathway in Post-Stroke Spasticity: A Diffusion Tensor Imaging Study.Journal of personalized medicine · 2021Article
- Corticoreticular Tract in the Human Brain: A Mini Review.Frontiers in neurology · 2019Review
Corrections and comments
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Authors and funding
2 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
objectivesWe report on a chronic hemiparetic patient whose gait recovery was delayed until healing of an injured corticoreticulospinal tract (CRT), which was demonstrated on diffusion tensor tractography (DTT). CASE PRESENTATION: A 71-year-old female presented with complete paralysis of the right extremities resulting from a spontaneous intracerebral hemorrhage. At 5 months after onset, when she was admitted for rehabilitation after undergoing rehabilitation at the previous university hospital, she presented with severe weakness of the right leg (manual muscle test: 0 ∼ 2- score) and could not even stand. She received comprehensive rehabilitative therapy for 32 months after the onset. Motor weakness of her right leg improved to the point that she was able to extend her knee on gravity-eliminated position at 11 months and against some resistance at 30 months after onset. She was able to walk independently at 30 months after onset.
resultsThe left CRT was discontinuous at the basal ganglia level on 5-month DTT. This discontinuation elongated to the cerebral cortex on 32-month DTT, whereas on 32-month DTT, the right CRT had become thicker compared with that on 5-month DTT.
conclusionsAn injured CRT healed in a patient who was able to walk independently after approximately 2 years of rehabilitation starting 5 months after the onset of intracerebral hemorrhage.
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Registered trials
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