Evidence map›Paper›PMID 27837018›Full record

ArticleJournal of leukocyte biology2017

Role of dendritic cell maturation factors produced by human invariant NKT cells in immune tolerance.

Hyeong-Woo Lee, Hyun Bae Jie, Paul L Bollyky, David Sarracino, Tong-Soo Kim, Brian S Wilson

Abstract read
In one paragraph

Article in Journal of leukocyte biology, 2017. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
–field-weighted citation impact, top 87% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed, 1 citations in OpenAlex.

  1. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors at 5 institutions in 2 countries.

Hyeong-Woo LeeDepartments of Tropical Medicine and Parasitology, Inha University School of Medicine, Incheon, Republic of Korea.
Hyun Bae JieOncoMed Pharmaceuticals, Inc., Redwood City, California.
Paul L BollykyDivision of Infectious Diseases, Stanford University Medical Center, Stanford, California; and.
David SarracinoThermo Fisher Scientific Biomarkers Research Initiatives in Mass Spectrometry (BRIMS) Center, Cambridge, Massachusetts.
Tong-Soo KimDepartments of Tropical Medicine and Parasitology, Inha University School of Medicine, Incheon, Republic of Korea; tongsookim@inha.ac.kr.
Brian S WilsonDepartment of Pathology, Immunology, and Laboratory Medicine, University of Florida, Gainesville, Florida; bwilson1@ufl.edu.
University of Florida · USInha University · KROncoMed (United States) · USStanford University · USThermo Fisher Scientific (United States) · US

Funding

Treatment of Autoimmune Disease by Costimulatory Signal*U19AI046130 · NIAID · BRIGHAM AND WOMEN'S HOSPITAL · PI KHOURY, SAMIA J. · 1999 to 2007
$5.6M
Regulation of iNKT and APC InteractionsR01AI045051 · NIAID · MASSACHUSETTS GENERAL HOSPITAL · PI WILSON, S. BRIAN BRIAN · 1999 to 2009
$2.7M
NIAID NIH HHS R01 AI045051NIAID NIH HHS U19 AI046130
6 · The paper itself

Abstract

In this study, we used the culture supernatant of iNKT cells to identify human myeloid DC maturation factors produced by human CD4

Indexed as

Immune ToleranceAntibodies, NeutralizingAntigens, CD1dCell DifferentiationCytokinesDendritic CellsHistocompatibility Antigens Class IIHumansModels, BiologicalNatural Killer T-CellsS100 ProteinsT-Lymphocytes, Helper-InducerT-Lymphocytes, RegulatoryToll-Like Receptor 4Transcription FactorsAntibodies, NeutralizingAntigens, CD1dCD1D protein, humanCytokinesHistocompatibility Antigens Class IIS100 ProteinsToll-Like Receptor 4Transcription FactorsATF-2CD4+ iNKT cellsHSF-1regulatory T cellTLR-4

Identifiers

PMID27837018
PMCPMC6608005
OpenAlexW2555070675

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.