Evidence map›Paper›PMID 27804968›Full record

ArticleHeredity2017

What causes mating system shifts in plants? Arabidopsis lyrata as a case study.

B K Mable, J Hagmann, S-T Kim, A Adam, E Kilbride, D Weigel, M Stift

Erratum issuedAbstract read
In one paragraph

Article in Heredity, 2017. The graph could read no effect estimate from its abstract, so it casts no vote on the map. An erratum has been issued. Cited by 23 papers.

0numbers the graph read from it
0cells of the map it votes in
23citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

23 citing papers in PubMed.

  1. Article
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  5. Genomic analyses elucidateEcology and evolution · 2024
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  9. International journal of molecular sciences · 2023
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  11. Article
  12. Review
  13. Article
  14. Article
  15. Article
  16. Article
  17. Population Genomics of Transitions to Selfing in Brassicaceae Model Systems.Methods in molecular biology (Clifton, N.J.) · 2020
    Article
  18. Article
  19. Article
  20. Article
4 · The record

Corrections and comments

5 · Who and what money

Authors and funding

7 authors.

B K MableInstitute of Biodiversity, Animal Health and Comparative Medicine, University of Glasgow, Glasgow, UK.
J HagmannDepartment of Molecular Biology, Max Planck Institute for Developmental Biology, Tübingen, Germany.
S-T KimDepartment of Molecular Biology, Max Planck Institute for Developmental Biology, Tübingen, Germany.
A AdamInstitute of Biodiversity, Animal Health and Comparative Medicine, University of Glasgow, Glasgow, UK.
E KilbrideInstitute of Biodiversity, Animal Health and Comparative Medicine, University of Glasgow, Glasgow, UK.
D WeigelDepartment of Molecular Biology, Max Planck Institute for Developmental Biology, Tübingen, Germany.
M StiftInstitute of Biodiversity, Animal Health and Comparative Medicine, University of Glasgow, Glasgow, UK.

Funding

Biotechnology and Biological Sciences Research Council BB/E024734/1
6 · The paper itself

Abstract

The genetic breakdown of self-incompatibility (SI) and subsequent mating system shifts to inbreeding has intrigued evolutionary geneticists for decades. Most of our knowledge is derived from interspecific comparisons between inbreeding species and their outcrossing relatives, where inferences may be confounded by secondary mutations that arose after the initial loss of SI. Here, we study an intraspecific breakdown of SI and its consequences in North American Arabidopsis lyrata to test whether: (1) particular S-locus haplotypes are associated with the loss of SI and/or the shift to inbreeding; (2) a population bottleneck may have played a role in driving the transition to inbreeding; and (3) the mutation(s) underlying the loss of SI are likely to have occurred at the S-locus. Combining multiple approaches for genotyping, we found that outcrossing populations on average harbour 5 to 9 S-locus receptor kinase (SRK) alleles, but only two, S1 and S19, are shared by most inbreeding populations. Self-compatibility (SC) behaved genetically as a recessive trait, as expected from a loss-of-function mutation. Bulked segregant analysis in SC × SI F2 individuals using deep sequencing confirmed that all SC plants were S1 homozygotes but not all S1 homozygotes were SC. This was also revealed in population surveys, where only a few S1 homozygotes were SC. Together with crossing data, this suggests that there is a recessive factor that causes SC that is physically unlinked to the S-locus. Overall, our results emphasise the value of combining classical genetics with advanced sequencing approaches to resolve long outstanding questions in evolutionary biology.

Indexed as

InbreedingArabidopsisBiological EvolutionGenes, PlantGenetics, PopulationGreat Lakes RegionHaplotypesPlant ProteinsPolymorphism, Single NucleotideProtein KinasesSelf-Incompatibility in Flowering PlantsPlant ProteinsProtein KinasesS-receptor kinase

Identifiers

PMID27804968
PMCPMC5176122

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.