Evidence map›Paper›PMID 27765917›Full record

ArticleOncotarget2016

Somatic alterations of targetable oncogenes are frequently observed in BRCA1/2 mutation negative male breast cancers.

Piera Rizzolo, Anna Sara Navazio, Valentina Silvestri, Virginia Valentini, Veronica Zelli, Ines Zanna, Giovanna Masala, Simonetta Bianchi, Marco Scarnò, Stefania Tommasi and 2 more

Open access · diamondAbstract read
In one paragraph

Article in Oncotarget, 2016. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 7 papers.

0numbers the graph read from it
0cells of the map it votes in
7citing papers in PubMed
1.2field-weighted citation impact, top 16% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

7 citing papers in PubMed, 9 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

12 authors at 4 institutions in 1 country.

Piera RizzoloDepartment of Molecular Medicine, Sapienza University of Rome, Rome, Italy.
Anna Sara NavazioDepartment of Molecular Medicine, Sapienza University of Rome, Rome, Italy.
Valentina SilvestriDepartment of Molecular Medicine, Sapienza University of Rome, Rome, Italy.
Virginia ValentiniDepartment of Molecular Medicine, Sapienza University of Rome, Rome, Italy.
Veronica ZelliDepartment of Molecular Medicine, Sapienza University of Rome, Rome, Italy.
Ines ZannaMolecular and Nutritional Epidemiology Unit, Cancer Research and Prevention Institute (ISPO), Florence, Italy.
Giovanna MasalaMolecular and Nutritional Epidemiology Unit, Cancer Research and Prevention Institute (ISPO), Florence, Italy.
Simonetta BianchiDivision of Pathological Anatomy, Department of Medical and Surgical Critical Care, University of Florence, Florence, Italy.
Marco ScarnòCINECA (Inter University Consortium for Super Computing), Rome, Italy.
Stefania TommasiMolecular Genetics Laboratory, Istituto Tumori "Giovanni Paolo II", Bari, Italy.
Domenico PalliMolecular and Nutritional Epidemiology Unit, Cancer Research and Prevention Institute (ISPO), Florence, Italy.
Laura OttiniDepartment of Molecular Medicine, Sapienza University of Rome, Rome, Italy.
Sapienza University of Rome · ITPiedmont Reference Center for Epidemiology and Cancer Prevention · ITIstituto Tumori Bari · ITUniversity of Florence · IT

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Male breast cancer (MBC) is a rare disease. Due to its rarity, MBC research and clinical approach are mostly based upon data derived from its largely known female counterpart. We aimed at investigating whether MBC cases harbor somatic alterations of genes known as prognostic biomarkers and molecular therapeutic targets in female breast cancer.We examined 103 MBC cases, all characterized for germ-line BRCA1/2 mutations, for somatic alterations in PIK3CA, EGFR, ESR1 and CCND1 genes.Pathogenic mutations of PIK3CA were detected in 2% of MBCs. No pathogenic mutations were identified in ESR1 and EGFR. Gene copy number variations (CNVs) analysis showed amplification of PIK3CA in 8.1%, EGFR in 6.8% and CCND1 in 16% of MBCs, whereas deletion of ESR1 was detected in 15% of MBCs. Somatic mutations and gene amplification were found only in BRCA1/2 mutation negative MBCs.Significant associations emerged between EGFR amplification and large tumor size (T4), ER-negative and HER2-positive status, between CCND1 amplification and HER2-positive and MIB1-positive status, and between ESR1 deletion and ER-negative status.Our results show that amplification of targetable oncogenes is frequent in BRCA1/2 mutation negative MBCs and may identify MBC subsets characterized by aggressive phenotype that may benefit from potential targeted therapeutic approaches.

Indexed as

Genes, BRCA1Genes, BRCA2Germ-Line MutationBase SequenceBreast Neoplasms, MaleClass I Phosphatidylinositol 3-KinasesDNA Copy Number VariationsGenetic Predisposition to DiseaseHumansMaleOncogenesClass I Phosphatidylinositol 3-KinasesPIK3CA protein, humangene copy number variationgerm-line BRCA1/2 mutationsmale breast canceroncogenessomatic mutations

Identifiers

PMID27765917
PMCPMC5342038
OpenAlexW2526743348

What OpenQuestion holds

Textmetadata
LicenceCC BY
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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.