ArticlePlant signaling & behavior2016
Role of Arabidopsis sterol 4α-methyl oxidase2 family in embryo and postembryonic development.
Article in Plant signaling & behavior, 2016. The graph could read no effect estimate from its abstract, so it casts no vote on the map. It has been retracted, and should not be counted. Cited by 1 paper.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
1 citing paper in PubMed, 2 citations in OpenAlex.
- Transcriptomic and Metabolomic Response to High Light in the Charophyte AlgaFrontiers in plant science · 2022Article
Corrections and comments
- Retraction · 2017-01-19Error in Results and/or Conclusions · Error in Text · Unreliable Results and/or Conclusions ·
- Retracted
Authors and funding
2 authors at 2 institutions in 1 country.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Sterols play important roles in plant growth, including embryogenesis, cell expansion, vascular differentiation, male fertility, and endocytosis. Sterols become functional only after removal of the 2 methyl groups at C-4. There are 2 distinct sterol C-4-methyl oxidase (SMO) families in higher plants, SMO1 and SMO2, which contain 3 and 2 isoforms, respectively, involving in the removal of the first and second C4 methyl groups during sterols biosynthesis. In a recent study we showed that single smo2-1 and smo2-2 mutants displayed no significant phenotype, while smo2-1 smo2-2 double mutant was embryonic lethal. smo2-1/+ smo2-2 and smo2-1 smo2-2/+ mutants showed defect in abnormal embryo patterning and smo2-1 smo2-2/+ mutant displayed dwarf phenotype. In this mini-review, we summarize the functions and regulatory mechanisms of SMO2-1 and SMO2-2 in embryo and postembryonic development.
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.