Why Is there a Limit to the Changes in Myofilament Ca

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

Who cites it

19 citing papers in PubMed, 24 citations in OpenAlex.

1. Troponin and a Myopathy-Linked Mutation inInternational journal of molecular sciences · 2023
   Article
2. Modulation of Structure and Dynamics of Cardiac Troponin by Phosphorylation and Mutations Revealed by Molecular Dynamics Simulations.The journal of physical chemistry. B · 2023
   Article
3. Recent studies of the molecular mechanism of lusitropy due to phosphorylation of cardiac troponin I by protein kinase A.Journal of muscle research and cell motility · 2023
   Review
4. A green tea extract catechin EGCg: Therapeutic potential for pediatric cardiomyopathies.Pediatric discovery · 2023
   Review
5. De Novo Asp219Val Mutation in Cardiac Tropomyosin Associated with Hypertrophic Cardiomyopathy.International journal of molecular sciences · 2022
   Article
6. Suppression of lusitropy as a disease mechanism in cardiomyopathies.Frontiers in cardiovascular medicine · 2022
   Review
7. Hypertrophic cardiomyopathy: Mutations to mechanisms to therapies.Frontiers in physiology · 2022
   Review
8. The R168G heterozygous mutation of tropomyosin 3 (TPM3) was identified in three family members and has manifestations ranging from asymptotic to serve scoliosis and respiratory complications.Genes & diseases · 2021
   Article
9. Looking for Targets to Restore the Contractile Function in Congenital Myopathy Caused by GlnInternational journal of molecular sciences · 2020
   Article
10. Troponin structure and function: a view of recent progress.Journal of muscle research and cell motility · 2020
    Review
11. Small molecule studies: the fourth wave of muscle research.Journal of muscle research and cell motility · 2019
    Article
12. Pathogenic troponin T mutants with opposing effects on myofilament CaArchives of biochemistry and biophysics · 2019
    Article
13. Cardiomyopathies and Related Changes in Contractility of Human Heart Muscle.International journal of molecular sciences · 2018
    Review
14. The Molecular Mechanisms of Mutations in Actin and Myosin that Cause Inherited Myopathy.International journal of molecular sciences · 2018
    Review
15. Age- and strain-related aberrant CaAmerican journal of physiology. Heart and circulatory physiology · 2017
    Article
16. The Myofilament Field Revisited in the Age of Cellular and Molecular Biology.Circulation research · 2017
    Review
17. Early sensitization of myofilaments to Ca2+ prevents genetically linked dilated cardiomyopathy in mice.Cardiovascular research · 2017
    Article
18. Cardiac contractility, motor function, and cross-bridge kinetics in N47K-RLC mutant mice.The FEBS journal · 2017
    Article
19. Investigations into the Sarcomeric Protein and CaFrontiers in physiology · 2017
    Article

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

Authors and funding

1 author at 1 institution in 1 country.

Funding

Abstract

Indexed as