Evidence map›Paper›PMID 27697884›Full record

ArticleASN neuro2016

Photoperiod Modulates Fast Delayed Rectifier Potassium Currents in the Mammalian Circadian Clock.

Sahar Farajnia, Johanna H Meijer, Stephan Michel

Open access · goldAbstract read
In one paragraph

Article in ASN neuro, 2016. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.

0numbers the graph read from it
0cells of the map it votes in
3citing papers in PubMed
0.9field-weighted citation impact, top 27% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

3 citing papers in PubMed, 9 citations in OpenAlex.

  1. Review
  2. Article
  3. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 1 institution in 1 country.

Sahar FarajniaLeiden University Medical Center, Leiden, The Netherlands Netherlands Institute for Neuroscience, Amsterdam, The Netherlands s.farajnia@nin.knaw.nl.
Johanna H MeijerLeiden University Medical Center, Leiden, The Netherlands.
Stephan MichelLeiden University Medical Center, Leiden, The Netherlands.
Leiden University Medical Center · NL

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

One feature of the mammalian circadian clock, situated in the suprachiasmatic nucleus (SCN), is its ability to measure day length and thereby contribute to the seasonal adaptation of physiology and behavior. The timing signal from the SCN, namely the 24 hr pattern of electrical activity, is adjusted according to the photoperiod being broader in long days and narrower in short days. Vasoactive intestinal peptide and gamma-aminobutyric acid play a crucial role in intercellular communication within the SCN and contribute to the seasonal changes in phase distribution. However, little is known about the underlying ionic mechanisms of synchronization. The present study was aimed to identify cellular mechanisms involved in seasonal encoding by the SCN. Mice were adapted to long-day (light-dark 16:8) and short-day (light-dark 8:16) photoperiods and membrane properties as well as K

Indexed as

PhotoperiodAnimalsBiophysicsDelayed Rectifier Potassium ChannelsDose-Response Relationship, DrugElectric StimulationIn Vitro TechniquesMaleMembrane PotentialsMiceMice, Inbred C57BLNeuronsPatch-Clamp TechniquesPotassium Channel BlockersSuprachiasmatic NucleusTetraethylammoniumDelayed Rectifier Potassium ChannelsPotassium Channel BlockersTetraethylammoniumcircadian rhythmsfast delayed rectifier currentphotoperiodpotassium channelsseasonalitysuprachiasmatic nucleus

Identifiers

PMID27697884
PMCPMC5051630
OpenAlexW2529371540

What OpenQuestion holds

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LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.