Evidence map›Paper›PMID 27695666›Full record

ArticleCurrent behavioral neuroscience reports2016

The Neurobiological Basis for Social Affiliation in Autism Spectrum Disorder and Schizophrenia.

Amanda Crider, Anilkumar Pillai

Abstract read
In one paragraph

Article in Current behavioral neuroscience reports, 2016. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed.

  1. Article
  2. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors.

Amanda CriderDepartment of Psychiatry and Health Behavior, Medical College of Georgia, Augusta University, Augusta, GA 30912.
Anilkumar PillaiDepartment of Psychiatry and Health Behavior, Medical College of Georgia, Augusta University, Augusta, GA 30912.

Funding

Regulation of TrkB signaling by 5-HT: role of transglutaminase 2R01MH097060 · NIMH · AUGUSTA UNIVERSITY · PI PILLAI, ANILKUMAR · 2012 to 2016
$1.5M
NIMH NIH HHS R01 MH097060United States NIMH
6 · The paper itself

Abstract

Social interaction and communication are complex behavioral paradigms involving many components. Many different neurotransmitters, hormones, sensory inputs, and brain regions are involved in the act of social engagement and verbal or nonverbal communication. Autism Spectrum Disorder (ASD) and schizophrenia are two neurodevelopmental disorders that have social and language deficits as hallmark symptoms, but show very different etiologies. The output of social dysfunction is common to both ASD and schizophrenia, but this likely arises from very different pathophysiological means. This review will attempt to compile and interpret human and animal studies showing the neurobiological basis for the development of social and language deficits in ASD and schizophrenia as well as a comparison of the two disorders.

Indexed as

autism spectrum disorderlanguage deficitsneurodevelopmental disordersschizophreniasocial affiliationsocial deficits

Identifiers

PMID27695666
PMCPMC5042200

What OpenQuestion holds

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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.