Evidence map›Paper›PMID 27690732›Full record

ArticlePlant signaling & behavior2016

"The roots" of selenium toxicity: A new concept.

Zsuzsanna Kolbert, Nóra Lehotai, Árpád Molnár, Gábor Feigl

Open access · bronzeAbstract read
In one paragraph

Article in Plant signaling & behavior, 2016. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 15 papers.

0numbers the graph read from it
0cells of the map it votes in
15citing papers in PubMed
2.2field-weighted citation impact, top 13% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

15 citing papers in PubMed, 55 citations in OpenAlex.

  1. Article
  2. Article
  3. An atlas of plant selenium metabolism.The New phytologist · 2026
    Review
  4. Review
  5. Toxic Effects of Acute Water Selenium Exposure onAnimals : an open access journal from MDPI · 2025
    Article
  6. Article
  7. Article
  8. Article
  9. Review
  10. The Defensive Role of Endogenous HInternational journal of molecular sciences · 2022
    Article
  11. Article
  12. Review
  13. Article
  14. Article
  15. Cytokinin at the Crossroads of Abiotic Stress Signalling Pathways.International journal of molecular sciences · 2018
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors at 2 institutions in 2 countries.

Zsuzsanna Kolberta Department of Plant Biology , University of Szeged , Szeged , Hungary.
Nóra Lehotaib Department of Plant Physiology , Umeå Plant Science Center, Umeå University , Umeå , Sweden.
Árpád Molnára Department of Plant Biology , University of Szeged , Szeged , Hungary.
Gábor Feigla Department of Plant Biology , University of Szeged , Szeged , Hungary.
University of Szeged · HUUmeå University · SE

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Elevated levels of selenium (Se) cause toxicity in non-accumulator plant species. The primary reasons for toxic Se effect have been considered to be selenoprotein accumulation and oxidative stress. However, based on our recent paper in Plant Cell Reports and previous literature data we suggest that disturbances in the homeostasis of both reactive oxygen and nitrogen species result in selenium-induced nitro-oxidative stress, contributing to toxicity. The most characteristic symptom of Se exposure is the inhibited root elongation which is partly caused by hormonal disturbances. Our recent paper suggests the involvement of cytokinin in selenium stress sensing of the root systemAltogether, the aim of this Addendum is to present reactive nitrogen species and phytohormones as new players in plant selenium toxicity.

Indexed as

CytokininsNitric OxideOxidative StressPlant RootsReactive Nitrogen SpeciesSeleniumCytokininsNitric OxideReactive Nitrogen SpeciesSeleniumCytokininnitric oxidephytohormonesreactive nitrogen speciesseleniumtoxicity

Identifiers

PMID27690732
PMCPMC5257165
OpenAlexW2528739643

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.