AP-1 is a key regulator of proinflammatory cytokine TNFα-mediated triple-negative breast cancer progression.

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

Who cites it

14 citing papers in PubMed.

1. Inflammation-associated drug resistance and tumor growth in TNBC.Frontiers in immunology · 2025
   Review
2. Surgical Tumor Resection Deregulates Hallmarks of Cancer in Resected Tissue and the Surrounding Microenvironment.Molecular cancer research : MCR · 2024
   Article
3. AP-1 is a regulatory transcription factor of inflammaging in the murine kidney and liver.Aging cell · 2023
   Article
4. Association between marital status and all-cause mortality of patients with metastatic breast cancer: a population-based study.Scientific reports · 2023
   Article
5. How cancer cells remodel lipid metabolism: strategies targeting transcription factors.Lipids in health and disease · 2021
   Review
6. Continuous Inflammatory Stimulation Leads via Metabolic Plasticity to a Prometastatic Phenotype in Triple-Negative Breast Cancer Cells.Cells · 2021
   Article
7. Transcription Factors in Cancer Development and Therapy.Cancers · 2020
   Review
8. The dual role of tumor necrosis factor-alpha (TNF-α) in breast cancer: molecular insights and therapeutic approaches.Cellular oncology (Dordrecht, Netherlands) · 2020
   Review
9. Timeless-Stimulated miR-5188-FOXO1/β-Catenin-c-Jun Feedback Loop Promotes Stemness via Ubiquitination of β-Catenin in Breast Cancer.Molecular therapy : the journal of the American Society of Gene Therapy · 2020
   Article
10. Transregulation of microRNA miR-21 promoter by AP-1 transcription factor in cervical cancer cells.Cancer cell international · 2019
    Article
11. Identification and bioinformatics analysis of overlapping differentially expressed genes in depression, papillary thyroid cancer and uterine fibroids.Experimental and therapeutic medicine · 2018
    Article
12. Hippo, TGF-β, and Src-MAPK pathways regulate transcription of the upd3 cytokine in Drosophila enterocytes upon bacterial infection.PLoS genetics · 2017
    Article
13. Prognostic value of increased expression of RACO-1 in patients with hepatitis B-related hepatocellular carcinoma.Therapeutics and clinical risk management · 2017
    Article
14. Flavonoids as Cytokine Modulators: A Possible Therapy for Inflammation-Related Diseases.International journal of molecular sciences · 2016
    Review

Corrections and comments

• Erratum for
  AP-1 Is a Key Regulator of Proinflammatory Cytokine TNFα-mediated Triple-negative Breast Cancer Progression.2016

Authors and funding

6 authors.

Funding

No grant is acknowledged in the PubMed record.

Abstract

PubMed holds no abstract for this paper.