Evidence map›Paper›PMID 27495161›Full record

ArticleCellular and molecular neurobiology2017

Nitric Oxide Upregulates Proteasomal Protein Degradation in Neurons.

Natalia Bal, Matvey Roshchin, Sergey Salozhin, Pavel Balaban

Open access · greenAbstract read
PubMed Full text at PMC Publisher
In one paragraph

Article in Cellular and molecular neurobiology, 2017. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
0.8field-weighted citation impact, top 25% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed, 9 citations in OpenAlex.

  1. Article
  2. Modulation of AMPA Receptors by Nitric Oxide in Nerve Cells.International journal of molecular sciences · 2020
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors at 1 institution in 1 country.

Natalia BalInstitute of Higher Nervous Activity and Neurophysiology of RAS, 5A Butlerova St., Moscow, 117485, Russia. bal_nv@mail.ru.
Matvey RoshchinInstitute of Higher Nervous Activity and Neurophysiology of RAS, 5A Butlerova St., Moscow, 117485, Russia.
Sergey SalozhinInstitute of Higher Nervous Activity and Neurophysiology of RAS, 5A Butlerova St., Moscow, 117485, Russia.
Pavel BalabanInstitute of Higher Nervous Activity and Neurophysiology of RAS, 5A Butlerova St., Moscow, 117485, Russia.
Institute of Higher Nervous Activity and Neurophysiology · RU

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Nitric oxide (NO) is involved in many neuronal functions such as neuromodulation and intracellular signaling. Recent studies have demonstrated that nitric oxide is involved in regulation of proteasomal protein degradation. However, its role in neuronal protein degradation still remains unclear. In our study, we investigated the influence of endogenous nitric oxide production in this process. We have shown that nitric oxide synthase blockade prevents decline of the Ub

Indexed as

AnimalsExcitatory Postsynaptic PotentialsFluorescenceGreen Fluorescent ProteinsHippocampusLong-Term PotentiationMaleNeuronsNitric OxideProteasome Endopeptidase ComplexProtein BiosynthesisProteolysisRats, WistarTime FactorsUbiquitinUp-RegulationGreen Fluorescent ProteinsNitric OxideProteasome Endopeptidase ComplexUbiquitinLong-term potentiationNeuronNitric oxideProteasomeProtein degradation

Identifiers

PMID27495161
PMCPMC11482057
OpenAlexW2499816633

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.