Evidence map›Paper›PMID 27493626›Full record

ArticleFrontiers in neuroanatomy2016

The Autophagoproteasome a Novel Cell Clearing Organelle in Baseline and Stimulated Conditions.

Paola Lenzi, Gloria Lazzeri, Francesca Biagioni, Carla L Busceti, Stefano Gambardella, Alessandra Salvetti, Francesco Fornai

Open access · goldAbstract read
In one paragraph

Article in Frontiers in neuroanatomy, 2016. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 40 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
40citing papers in PubMed, 1 pooled it
3.3field-weighted citation impact, top 8% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

40 citing papers in PubMed, 1 synthesis or guideline pooled it, 44 citations in OpenAlex.

  1. Guideline
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors at 3 institutions in 1 country.

Paola LenziDepartment of Translational Research and New Technologies in Medicine and Surgery, University of Pisa Pisa, Italy.
Gloria LazzeriDepartment of Translational Research and New Technologies in Medicine and Surgery, University of Pisa Pisa, Italy.
Francesca BiagioniIstituti di Ricovero e Cura a Carattere Scientifico (I.R.C.C.S.), Neuromed Pozzilli, Italy.
Carla L BuscetiIstituti di Ricovero e Cura a Carattere Scientifico (I.R.C.C.S.), Neuromed Pozzilli, Italy.
Stefano GambardellaIstituti di Ricovero e Cura a Carattere Scientifico (I.R.C.C.S.), Neuromed Pozzilli, Italy.
Alessandra SalvettiDepartment of Clinical and Experimental Medicine, University of Pisa Pisa, Italy.
Francesco FornaiDepartment of Translational Research and New Technologies in Medicine and Surgery, University of PisaPisa, Italy; Istituti di Ricovero e Cura a Carattere Scientifico (I.R.C.C.S.), NeuromedPozzilli, Italy.
Istituto Neurologico Mediterraneo · ITUniversity of Pisa · ITIstituti di Ricovero e Cura a Carattere Scientifico · IT

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Protein clearing pathways named autophagy (ATG) and ubiquitin proteasome (UP) control homeostasis within eukaryotic cells, while their dysfunction produces neurodegeneration. These pathways are viewed as distinct biochemical cascades occurring within specific cytosolic compartments owing pathway-specific enzymatic activity. Recent data strongly challenged the concept of two morphologically distinct and functionally segregated compartments. In fact, preliminary evidence suggests the convergence of these pathways to form a novel organelle named autophagoproteasome. This is characterized in the present study by using a cell line where, mTOR activity is upregulated and autophagy is suppressed. This was reversed dose-dependently by administering the mTOR inhibitor rapamycin. Thus, we could study autophagoproteasomes when autophagy was either suppressed or stimulated. The occurrence of autophagoproteasome was shown also in non-human cell lines. Ultrastructural morphometry, based on the stochiometric binding of immunogold particles allowed the quantitative evaluation of ATG and UP component within autophagoproteasomes. The number of autophagoproteasomes increases following mTOR inhibition. Similarly, mTOR inhibition produces overexpression of both LC3 and P20S particles. This is confirmed by the fact that the ratio of free vs. autophagosome-bound LC3 is similar to that measured for P20S, both in baseline conditions and following mTOR inhibition. Remarkably, within autophagoproteasomes there is a slight prevalence of ATG compared with UP components for low rapamycin doses, whereas for higher rapamycin doses UP increases more than ATG. While LC3 is widely present within cytosol, UP is strongly polarized within autophagoproteasomes. These fine details were evident at electron microscopy but could not be deciphered by using confocal microscopy. Despite its morphological novelty autophagoproteasomes appear in the natural site where clearing pathways (once believed to be anatomically segregated) co-exist and they are likely to interact at molecular level. In fact, LC3 and P20S co-immunoprecipitate, suggesting a specific binding and functional interplay, which may be altered by inhibiting mTOR. In summary, ATG and UP often represent two facets of a single organelle, in which unexpected amount of enzymatic activity should be available. Thus, autophagoproteasome may represent a sophisticated ultimate clearing apparatus.

Indexed as

autophagosomeautophagyproteasomeprotein-clearing-pathwaysubiquitination

Identifiers

PMID27493626
PMCPMC4955296
OpenAlexW2488631391

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.