Evidence map›Paper›PMID 27490094›Full record

ArticleChemical research in toxicology2016

Translesion Synthesis of the N(2)-2'-Deoxyguanosine Adduct of the Dietary Mutagen IQ in Human Cells: Error-Free Replication by DNA Polymerase κ and Mutagenic Bypass by DNA Polymerases η, ζ, and Rev1.

Arindam Bose, Amy D Millsap, Arnie DeLeon, Carmelo J Rizzo, Ashis K Basu

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Article in Chemical research in toxicology, 2016. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.

0numbers the graph read from it
0cells of the map it votes in
4citing papers in PubMed
0.9field-weighted citation impact, top 23% of its field
1 · What the graph read from it

What it found

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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

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3 · Its place in the literature

Who cites it

4 citing papers in PubMed, 12 citations in OpenAlex.

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4 · The record

Corrections and comments

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5 · Who and what money

Authors and funding

5 authors at 2 institutions in 1 country.

Arindam BoseDepartment of Chemistry, University of Connecticut , Storrs, Connecticut 06269, United States.
Amy D MillsapDepartment of Chemistry, Vanderbilt University , Nashville, Tennessee 37232, United States.
Arnie DeLeonDepartment of Chemistry, Vanderbilt University , Nashville, Tennessee 37232, United States.
Carmelo J RizzoDepartment of Chemistry, Vanderbilt University , Nashville, Tennessee 37232, United States.
Ashis K BasuDepartment of Chemistry, University of Connecticut , Storrs, Connecticut 06269, United States.
Vanderbilt University · USUniversity of Connecticut · US

Funding

Tumor Immunology and Microenvironment Research ProgramP30CA068485 · NCI · VANDERBILT UNIVERSITY MEDICAL CENTER · PI STACIE B DUSETZINA · 1995 to 2026
$172.8M
Structural Biology Facility CoreP30ES000267 · NIEHS · VANDERBILT UNIVERSITY · PI GUENGERICH, F PETER · 1985 to 2014
$20.3M
NCI NIH HHS P30 CA068485NIEHS NIH HHS P30 ES000267
6 · The paper itself

Abstract

Translesion synthesis (TLS) of the N(2)-2'-deoxyguanosine (dG-N(2)-IQ) adduct of the carcinogen 2-amino-3-methylimidazo[4,5-f]quinoline (IQ) was investigated in human embryonic kidney 293T cells by replicating plasmid constructs in which the adduct was individually placed at each guanine (G1, G2, or G3) of the NarI sequence (5'-CG1G2CG3CC-3'). TLS efficiency was 38%, 29%, and 25% for the dG-N(2)-IQ located at G1, G2, and G3, respectively, which suggests that dG-N(2)-IQ is bypassed more efficiently by one or more DNA polymerases at G1 than at either G2 or G3. TLS efficiency was decreased 8-35% in cells with knockdown of pol η, pol κ, pol ι, pol ζ, or Rev1. Up to 75% reduction in TLS occurred when pol η, pol ζ, and Rev1 were simultaneously knocked down, suggesting that these three polymerases play important roles in dG-N(2)-IQ bypass. Mutation frequencies (MFs) of dG-N(2)-IQ at G1, G2, and G3 were 23%, 17%, and 11%, respectively, exhibiting a completely reverse trend of the previously reported MF of the C8-dG adduct of IQ (dG-C8-IQ), which is most mutagenic at G3 ( ( 2015 ) Nucleic Acids Res. 43 , 8340 - 8351 ). The major type of mutation induced by dG-N(2)-IQ was targeted G → T, as was reported for dG-C8-IQ. In each site, knockdown of pol κ resulted in an increase in MF, whereas MF was reduced when pol η, pol ι, pol ζ, or Rev1 was knocked down. The reduction in MF was most pronounced when pol η, pol ζ, and Rev1 were simultaneously knocked down and especially when the adduct was located at G3, where MF was reduced by 90%. We conclude that pol κ predominantly performs error-free TLS of the dG-N(2)-IQ adduct, whereas pols η, pol ζ, and Rev1 cooperatively carry out the error-prone TLS. However, in vitro experiments using yeast pol ζ and κ showed that the former was inefficient in full-length primer extension on dG-N(2)-IQ templates, whereas the latter was efficient in both error-free and error-prone extensions. We believe that the observed differences between the in vitro experiments using purified DNA polymerases, and the cellular results may arise from several factors including the crucial roles played by the accessory proteins in TLS.

Indexed as

DietDeoxyguanosineDNA AdductsDNA-Directed DNA PolymeraseDNA Polymerase iotaDNA ReplicationHEK293 CellsHumansImidazolesIsoleucineMolecular StructureMutagensNuclear ProteinsNucleotidyltransferasesQuinoxalinesY-Family DNA Polymerases2-amino-3-methylpentanoic acidDeoxyguanosineDNA AdductsDNA-Directed DNA PolymeraseDNA Polymerase iotaimidazo(1,2-a)quinoxalin-4-amineImidazolesIsoleucineMutagensNuclear ProteinsNucleotidyltransferasesPOLI protein, humanQuinoxalinesREV1 protein, humanY-Family DNA Polymerases

Identifiers

PMID27490094
PMCPMC5031085
OpenAlexW2490251124

What OpenQuestion holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.