Evidence map›Paper›PMID 27481636›Full record

ArticleHaemophilia : the official journal of the World Federation of Hemophilia2016

In silico analysis highlights the copy number variation mechanism responsible for the historically reported VWF exon 42 deletion.

A Cartwright, I R Peake, A C Goodeve, D J Hampshire

Open access · greenAbstract readCase ReportsLetter
PubMed Full text at PMC Publisher
In one paragraph

Article in Haemophilia : the official journal of the World Federation of Hemophilia, 2016. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact, top 94% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed, 0 citations in OpenAlex.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors at 1 institution in 1 country.

A CartwrightHaemostasis Research Group, Department of Infection, Immunity and Cardiovascular Disease, University of Sheffield, Sheffield, UK.
I R PeakeHaemostasis Research Group, Department of Infection, Immunity and Cardiovascular Disease, University of Sheffield, Sheffield, UK.
A C GoodeveHaemostasis Research Group, Department of Infection, Immunity and Cardiovascular Disease, University of Sheffield, Sheffield, UK.ORCID http://orcid.org/0000-0002-6177-7062
D J HampshireHaemostasis Research Group, Department of Infection, Immunity and Cardiovascular Disease, University of Sheffield, Sheffield, UK. d.hampshire@shef.ac.uk.ORCID http://orcid.org/0000-0002-1387-8926
University of Sheffield · GB

Funding

Zimmerman Program for the Molecular and Clinical Biology of VWDP01HL081588 · NHLBI · VERSITI WISCONSIN, INC. · PI MONTGOMERY, ROBERT R · 2005 to 2016
$19.3M
NHLBI NIH HHS P01 HL081588
6 · The paper itself

Abstract

PubMed holds no abstract for this paper.

Indexed as

Base SequenceDNA Copy Number VariationsExonsHumansMolecular Sequence DataPolymorphism, Single NucleotideSequence AlignmentSequence Deletionvon Willebrand Factorvon Willebrand Factor

Identifiers

PMID27481636
PMCPMC5960579
OpenAlexW2488419658

What OpenQuestion holds

Textmetadata
LicenceTDM
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.