Evidence map›Paper›PMID 27445717›Full record

ReviewFrontiers in systems neuroscience2016

Behavioral Consequences and Cortical Reorganization in Homonymous Hemianopia.

Sylvie Chokron, Céline Perez, Carole Peyrin

Open access · goldAbstract readReview
In one paragraph

Review in Frontiers in systems neuroscience, 2016. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 11 papers.

0numbers the graph read from it
0cells of the map it votes in
11citing papers in PubMed
2.5field-weighted citation impact, top 11% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

11 citing papers in PubMed, 28 citations in OpenAlex.

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  11. Neurobiology as Information Physics.Frontiers in systems neuroscience · 2016
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 2 institutions in 1 country.

Sylvie ChokronUnité Fonctionnelle Vision and Cognition, Fondation Ophtalmologique RothschildParis, France; UMR 8242, Laboratoire de Psychologie de la Perception, CNRS and Université Paris-DescartesParis, France.
Céline PerezUnité Fonctionnelle Vision and Cognition, Fondation Ophtalmologique RothschildParis, France; UMR 8242, Laboratoire de Psychologie de la Perception, CNRS and Université Paris-DescartesParis, France.
Carole PeyrinUMR 5105, CNRS, Laboratoire de Psychologie et NeuroCognition, Université Grenoble Alpes Grenoble, France.
Laboratoire Psychologie de la Perception · FRLaboratoire de Psychologie et NeuroCognition · FR

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The most common visual defect to follow a lesion of the retrochiasmal pathways is homonymous hemianopia (HH), whereby, in each eye, patients are blind to the contralesional visual field. From a behavioral perspective, in addition to exhibiting a severe deficit in their contralesional visual field, hemianopic patients can also present implicit residual capacities, now usually referred to collectively as blindsight. It was recently demonstrated that HH patients can also suffer from a subtle deficit in their ipsilesional visual field, called sightblindness (the reverse case of blindsight). Furthermore, the nature of the visual deficit in the contralesional and ipsilesional visual fields, as well as the pattern of functional reorganization in the occipital lobe of HH patients after stroke, all appear to depend on the lesion side. In addition to their contralesional and ipsilesional visual deficits, and to their residual capacities, HH patients can also experience visual hallucinations in their blind field, the physiopathological mechanisms of which remain poorly understood. Herein we review blindsight in terms of its better-known aspects as well as its less-studied clinical signs such as sightblindness, hemispheric specialization and visual hallucinations. We also discuss the implications of recent experimental findings for rehabilitation of visual field defects in hemianopic patients.

Indexed as

blindsighthallucinationshomonymous hemianopiaoccipital lobesightblindness

Identifiers

PMID27445717
PMCPMC4923162
OpenAlexW2461966313

What OpenQuestion holds

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LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.