Evidence map›Paper›PMID 27390209›Full record

ReviewCirculation. Arrhythmia and electrophysiology2016

Calcium Revisited: New Insights Into the Molecular Basis of Long-QT Syndrome.

John R Giudicessi, Michael J Ackerman

Abstract readReview
PubMed Full text at PMC Publisher
In one paragraph

Review in Circulation. Arrhythmia and electrophysiology, 2016. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 10 papers.

0numbers the graph read from it
0cells of the map it votes in
10citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

10 citing papers in PubMed.

  1. Article
  2. Article
  3. Article
  4. Article
  5. Article
  6. Voltage-Gated CaFrontiers in synaptic neuroscience · 2021
    Review
  7. Review
  8. Review
  9. Long QT Syndrome and Sinus Bradycardia-A Mini Review.Frontiers in cardiovascular medicine · 2018
    Review
  10. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors.

John R GiudicessiFrom the Internal Medicine Residency and Clinician-Investigator Programs, Department of Medicine (J.R.G.) and Departments of Cardiovascular Diseases, Pediatrics (Division of Pediatric Cardiology), and Molecular Pharmacology & Experimental Therapeutics (M.J.A.), Windland Smith Rice Sudden Death Genomics Laboratory, Mayo Clinic, Rochester, MN.
Michael J AckermanFrom the Internal Medicine Residency and Clinician-Investigator Programs, Department of Medicine (J.R.G.) and Departments of Cardiovascular Diseases, Pediatrics (Division of Pediatric Cardiology), and Molecular Pharmacology & Experimental Therapeutics (M.J.A.), Windland Smith Rice Sudden Death Genomics Laboratory, Mayo Clinic, Rochester, MN. ackerman.michael@mayo.edu.

Funding

Cardiac Channel Mutations in Sudden Infant Death Syndrome (SIDS)R01HD042569 · NICHD · MAYO CLINIC ROCHESTER · PI ACKERMAN, MICHAEL JOHN · 2002 to 2017
$6.9M
NICHD NIH HHS R01 HD042569
6 · The paper itself

Abstract

PubMed holds no abstract for this paper.

Indexed as

CalciumCalcium ChannelsCalcium Channels, L-TypeCalmodulinCarrier ProteinsElectrocardiographyGenetic Predisposition to DiseaseGenetic VariationGenotypeHumansLong QT SyndromeMuscle ProteinsMutationPhenotypeRyanodine Receptor Calcium Release ChannelCACNA1C protein, humanCalciumCalcium ChannelsCalcium Channels, L-TypeCALM1 protein, humanCalmodulinCarrier ProteinsMuscle ProteinsRyanodine Receptor Calcium Release ChannelRyR2 protein, humanTRDN protein, humancalciumhumanion channellong-QT syndromesudden cardiac death

Identifiers

PMID27390209
PMCPMC6364983

What OpenQuestion holds

Textmetadata
LicenceTDM
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.