Evidence map›Paper›PMID 27375018›Full record

ArticleOncogene2017

A large-scale RNAi screen identifies LCMR1 as a critical regulator of Tspan8-mediated melanoma invasion.

G Agaësse, L Barbollat-Boutrand, E Sulpice, R Bhajun, M El Kharbili, O Berthier-Vergnes, F Degoul, A de la Fouchardière, E Berger, T Voeltzel and 3 more

Erratum issuedAbstract read
PubMed Publisher
In one paragraph

Article in Oncogene, 2017. The graph could read no effect estimate from its abstract, so it casts no vote on the map. An erratum has been issued. Cited by 14 papers.

0numbers the graph read from it
0cells of the map it votes in
14citing papers in PubMed
1.1field-weighted citation impact, top 23% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

14 citing papers in PubMed, 31 citations in OpenAlex.

  1. Review
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  4. Deletion ofJournal of thoracic disease · 2023
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4 · The record

Corrections and comments

5 · Who and what money

Authors and funding

13 authors at 7 institutions in 1 country.

G AgaësseUniversité de Lyon, Lyon, France.
L Barbollat-BoutrandUniversité de Lyon, Lyon, France.
E SulpiceUniversité Grenoble-Alpes, Grenoble, France.
R BhajunUniversité Grenoble-Alpes, Grenoble, France.
M El KharbiliUniversité de Lyon, Lyon, France.
O Berthier-VergnesUniversité de Lyon, Lyon, France.
F DegoulClermont Université, Université d'Auvergne, Imagerie Moléculaire et Thérapie Vectorisée, BP, Clermont-Ferrand, France.
A de la FouchardièreDépartement de Biopathologie, Centre Léon Bérard, Lyon, France.
E BergerLaboratoire CarMeN (INSERM 1060, INRA 1397, INSA), Université de Lyon, Lyon, France.
T VoeltzelCentre de Recherche en Cancérologie de Lyon, CNRS UMR5286, Inserm U1052, Université de Lyon, Université Lyon 1, Lyon, France.
J LamartineUniversité de Lyon, Lyon, France.
X GidrolUniversité Grenoble-Alpes, Grenoble, France.
I MasseUniversité de Lyon, Lyon, France.
Université Claude Bernard Lyon 1 · FRCentre National de la Recherche Scientifique · FRInserm · FRCentre de Recherche en Cancérologie de Lyon · FRCentre Léon Bérard · FRLaboratoire Biologie à Grande Échelle · FRUniversité Clermont Auvergne · FR

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Melanoma is the deadliest form of skin cancer owing to its proclivity to metastasise, and recently developed therapies have not yielded the expected results, because almost all patients relapse. Therefore, understanding the molecular mechanisms that underlie early invasion by melanoma cells is crucial to improving patient survival. We have previously shown that, whereas the Tetraspanin 8 protein (Tspan8) is undetectable in normal skin and benign lesions, its expression arises with the progression of melanoma and is sufficient to increase cell invasiveness. Therefore, to identify Tspan8 transcriptional regulators that could explain the onset of Tspan8 expression, thereby conferring an invasive phenotype, we performed an innovative RNA interference-based screen, which, for the first time, identified several Tspan8 repressors and activators, such as GSK3β, PTEN, IQGAP1, TPT1 and LCMR1. LCMR1 is a recently identified protein that is overexpressed in numerous carcinomas; its expression and role, however, had not previously been studied in melanoma. The present study identified Tspan8 as the first LCMR1 target that could explain its function in carcinogenesis. LCMR1 modulation was sufficient to positively regulate endogenous Tspan8 expression, with concomitant in vitro phenotypic changes such as loss of melanoma cell-matrix adherence and increase in invasion, and Tspan8 expression promoted tumourigenicity in vivo. Moreover, LCMR1 and Tspan8 overexpression were shown to correlate in melanoma lesions, and both proteins could be downregulated in vitro by vemurafenib. In conclusion, this study highlights the importance of Tspan8 and its regulators in the control of early melanoma invasion and suggests that they may be promising new therapeutic targets downstream of the RAF-MEK-ERK signalling pathway.

Indexed as

AnimalsCell Line, TumorHeterograftsHumansMaleMediator ComplexMelanomaMiceMice, NudeNeoplasm InvasivenessRNA InterferenceSignal TransductionSkin NeoplasmsTetraspaninsTransfectionTumor Protein, Translationally-Controlled 1MED19 protein, humanMediator ComplexTetraspaninsTPT1 protein, humanTpt1 protein, mouseTSPAN8 protein, humanTumor Protein, Translationally-Controlled 1

Identifiers

PMID27375018
OpenAlexW2466380273

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.