Genome-wide association studies identify genetic loci for low von Willebrand factor levels.

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

Who cites it

6 citing papers in PubMed.

1. Von Willebrand disease: A century of progress.Research and practice in thrombosis and haemostasis · 2026
   Review
2. RAB5C Increases Endothelial Release of VWF by Regulating Vesicle Trafficking.Arteriosclerosis, thrombosis, and vascular biology · 2026
   Article
3. Analysis of von Willebrand Disease in the "Heart of Europe".TH open : companion journal to thrombosis and haemostasis · 2022
   Article
4. Molecular pathogenesis and heterogeneity in type 3 VWD families in U.S. Zimmerman program.Journal of thrombosis and haemostasis : JTH · 2022
   Article
5. Regulation of plasma von Willebrand factor.F1000Research · 2018
   Review
6. Von Willebrand disease in the elderly: clinical perspectives.Clinical interventions in aging · 2018
   Review

Corrections and comments

• Erratum for
  Genome-wide association studies identify genetic loci for low von Willebrand factor levels.2016

Authors and funding

33 authors.

Funding

Abstract

PubMed holds no abstract for this paper.