Evidence map›Paper›PMID 27288076›Full record

ReviewAdvances in pharmacology (San Diego, Calif.)2016

Transcriptional Regulation of Glutamate Transporters: From Extracellular Signals to Transcription Factors.

Z Martinez-Lozada, A M Guillem, M B Robinson

Abstract readReview
In one paragraph

Review in Advances in pharmacology (San Diego, Calif.), 2016. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 43 papers.

0numbers the graph read from it
0cells of the map it votes in
43citing papers in PubMed
8.2field-weighted citation impact, top 2% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

43 citing papers in PubMed, 65 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 2 institutions in 1 country.

Z Martinez-LozadaChildren's Hospital of Philadelphia Research Institute, University of Pennsylvania, Philadelphia, PA, United States.
A M GuillemChildren's Hospital of Philadelphia Research Institute, University of Pennsylvania, Philadelphia, PA, United States.
M B RobinsonChildren's Hospital of Philadelphia Research Institute, University of Pennsylvania, Philadelphia, PA, United States. Electronic address: robinson@mail.med.upenn.edu.
Children's Hospital of Philadelphia · USUniversity of Pennsylvania · US

Funding

Regulation of glutamate transport in astrocyte subtypes and in ALSR01NS092067 · NINDS · CHILDREN'S HOSP OF PHILADELPHIA · PI ROBINSON, MICHAEL BYRNE, ROTHSTEIN, JEFFREY D · 2015 to 2019
$2.3M
NINDS NIH HHS R01 NS092067
6 · The paper itself

Abstract

Glutamate is the predominant excitatory neurotransmitter in the mammalian CNS. It mediates essentially all rapid excitatory signaling. Dysfunction of glutamatergic signaling contributes to developmental, neurologic, and psychiatric diseases. Extracellular glutamate is cleared by a family of five Na(+)-dependent glutamate transporters. Two of these transporters (GLAST and GLT-1) are relatively selectively expressed in astrocytes. Other of these transporters (EAAC1) is expressed by neurons throughout the nervous system. Expression of the last two members of this family (EAAT4 and EAAT5) is almost exclusively restricted to specific populations of neurons in cerebellum and retina, respectively. In this review, we will discuss our current understanding of the mechanisms that control transcriptional regulation of the different members of this family. Over the last two decades, our understanding of the mechanisms that regulate expression of GLT-1 and GLAST has advanced considerably; several specific transcription factors, cis-elements, and epigenetic mechanisms have been identified. For the other members of the family, little or nothing is known about the mechanisms that control their transcription. It is assumed that by defining the mechanisms involved, we will advance our understanding of the events that result in cell-specific expression of these transporters and perhaps begin to define the mechanisms by which neurologic diseases are changing the biology of the cells that express these transporters. This approach might provide a pathway for developing new therapies for a wide range of essentially untreatable and devastating diseases that kill neurons by an excitotoxic mechanism.

Indexed as

Amino Acid Transport System X-AGAnimalsAstrocytesATP-Binding Cassette TransportersGlutamic AcidHumansNeuronsSignal TransductionTranscription FactorsAmino Acid Transport System X-AGATP-Binding Cassette TransportersGlutamic AcidTranscription FactorsAstrocytesEAAC1EAATGLASTGLT-1Glu uptakeTranscriptional regulation

Identifiers

PMID27288076
PMCPMC5544923
OpenAlexW2346518875

What OpenQuestion holds

Textmetadata
LicenceTDM
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.