Evidence map›Paper›PMID 27253400›Full record

ArticlePloS one2016

Finasteride Enhances the Generation of Human Myeloid-Derived Suppressor Cells by Up-Regulating the COX2/PGE2 Pathway.

Shaoying Zhang, Kang Wu, Yufeng Liu, Yingtong Lin, Xu Zhang, Jie Zhou, Hui Zhang, Ting Pan, Yongshui Fu

Open access · goldAbstract read
In one paragraph

Article in PloS one, 2016. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 8 papers.

0numbers the graph read from it
0cells of the map it votes in
8citing papers in PubMed
0.6field-weighted citation impact, top 31% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

8 citing papers in PubMed, 11 citations in OpenAlex.

  1. Article
  2. Article
  3. Review
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  7. MDSCs in infectious diseases: regulation, roles, and readjustment.Cancer immunology, immunotherapy : CII · 2019
    Review
  8. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors at 3 institutions in 1 country.

Shaoying ZhangInstitute of Human Virology, Sun Yat-sen University, Guangzhou, Guangdong, China.
Kang WuInstitute of Human Virology, Sun Yat-sen University, Guangzhou, Guangdong, China.
Yufeng LiuInstitute of Human Virology, Sun Yat-sen University, Guangzhou, Guangdong, China.
Yingtong LinInstitute of Human Virology, Sun Yat-sen University, Guangzhou, Guangdong, China.
Xu ZhangInstitute of Human Virology, Sun Yat-sen University, Guangzhou, Guangdong, China.
Jie ZhouInstitute of Human Virology, Sun Yat-sen University, Guangzhou, Guangdong, China.
Hui ZhangInstitute of Human Virology, Sun Yat-sen University, Guangzhou, Guangdong, China.
Ting PanInstitute of Human Virology, Sun Yat-sen University, Guangzhou, Guangdong, China.
Yongshui FuGuangzhou Blood Center, Guangzhou, Guangdong, China.
Sun Yat-sen University · CNGuangzhou Blood Center · CNGuangzhou Medical University · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Myeloid-derived suppressor cells (MDSCs) have been known to be a key factor in the regulation of the immune system under numerous conditions such as tumors, infections, autoimmune diseases, and transplantations. In contrast to the proposed deleterious role of MDSCs in tumors and infections, MDSCs with their suppressive function are now proved to have the beneficial potential of suppressing the autoimmune response and promoting tolerance to transplantation. Therefore, the expansion of MDSCs could be a promising therapeutic strategy for many diseases. In this study, we aimed to identify FDA-approved drugs that could aid in the expansion of functional MDSCs. We performed a high-throughput screening (HTS) of FDA-approved drugs based on the in vitro human MDSC-differentiation system and identified finasteride (FIN) to have the best potency to aid the generation of human MDSCs. The FIN-induced MDSCs were quite similar to monocytic MDSCs with regard to their surface phenotype, morphology, immunosuppressive function, and related gene expression. Next, we aimed to determine the mechanism of action of FIN and found that FIN induced the expansion of MDSCs through up-regulation of the COX2/PGE2 pathway by enhancing the activity of COX2 promoter. In addition, the administration of indomethacin (IND), a COX2 inhibitor, abrogated the effect of FIN. Based on these results, we suggested that FIN could find applications in the future in the expansion of MDSCs. Further development of FIN-like compounds could be a novel strategy for generating functional MDSCs for immunosuppressive therapies in various immune disorder conditions.

Indexed as

AutoimmunityCell LineCyclooxygenase 2Cyclooxygenase 2 InhibitorsFinasterideHumansImmune System DiseasesImmune ToleranceIndomethacinMyeloid-Derived Suppressor CellsNeoplasmsSignal TransductionCyclooxygenase 2Cyclooxygenase 2 InhibitorsFinasterideIndomethacin

Identifiers

PMID27253400
PMCPMC4890941
OpenAlexW2411327930

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.