Evidence map›Paper›PMID 27173378›Full record

ArticleJournal of molecular biology2016

POT1-TPP1 Binding and Unfolding of Telomere DNA Discriminates against Structural Polymorphism.

Michael R Mullins, Malligarjunan Rajavel, Wilnelly Hernandez-Sanchez, Maria de la Fuente, Sherri M Biendarra, Michael E Harris, Derek J Taylor

Abstract read
In one paragraph

Article in Journal of molecular biology, 2016. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 17 papers.

0numbers the graph read from it
0cells of the map it votes in
17citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

17 citing papers in PubMed.

  1. Article
  2. Article
  3. Article
  4. Article
  5. Article
  6. Article
  7. Review
  8. G-Quadruplexes at Telomeres: Friend or Foe?Molecules (Basel, Switzerland) · 2020
    Review
  9. Article
  10. Article
  11. POT1-TPP1 differentially regulates telomerase via POT1 His266 and as a function of single-stranded telomere DNA length.Proceedings of the National Academy of Sciences of the United States of America · 2019
    Article
  12. Review
  13. Article
  14. Article
  15. Article
  16. Article
  17. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors.

Michael R MullinsDepartment of Pharmacology, Case Western Reserve University, Cleveland, OH 44106, USA.
Malligarjunan RajavelDepartment of Pharmacology, Case Western Reserve University, Cleveland, OH 44106, USA.
Wilnelly Hernandez-SanchezDepartment of Pharmacology, Case Western Reserve University, Cleveland, OH 44106, USA.
Maria de la FuenteDepartment of Pharmacology, Case Western Reserve University, Cleveland, OH 44106, USA.
Sherri M BiendarraDepartment of Pharmacology, Case Western Reserve University, Cleveland, OH 44106, USA.
Michael E HarrisDepartment of Biochemistry, Case Western Reserve University, Cleveland, OH 44106, USA.
Derek J TaylorDepartment of Pharmacology, Case Western Reserve University, Cleveland, OH 44106, USA; Department of Biochemistry, Case Western Reserve University, Cleveland, OH 44106, USA. Electronic address: derek.taylor@case.edu.

Funding

STRUCTURE/FUNCTION OF RIBONUCLEASE PR01GM056740 · NIGMS · CASE WESTERN RESERVE UNIVERSITY · PI HARRIS, MICHAEL E. · 1998 to 2015
$4.8M
Induction of cancer cell death by selective DNA misincorporationDP2CA186571 · NCI · CASE WESTERN RESERVE UNIVERSITY · PI TAYLOR, DEREK JAMES · 2013 to 2014
$2.6M
Predoctoral Training Program in Molecular PharmacologyT32GM072474 · NIGMS · MAYO CLINIC ROCHESTER · PI KARNITZ, LARRY M · 2005 to 2020
$2.4M
NCI NIH HHS DP2 CA186571NIGMS NIH HHS R01 GM056740NIGMS NIH HHS T32 GM072474
6 · The paper itself

Abstract

Telomeres are nucleoprotein complexes that reside at the ends of linear chromosomes and help maintain genomic integrity. Protection of telomeres 1 (POT1) and TPP1 are telomere-specific proteins that bind as a heterodimer to single-stranded telomere DNA to prevent illicit DNA damage responses and to enhance telomerase-mediated telomere extension. Telomere DNA is guanosine rich and, as such, can form highly stable secondary structures including G-quadruplexes. G-quadruplex DNA folds into different topologies that are determined by several factors including monovalent ion composition and the precise sequence and length of the DNA. Here, we explore the influence of DNA secondary structure on POT1-TPP1 binding. Equilibrium binding assays reveal that the POT1-TPP1 complex binds G-quadruplex structures formed in buffers containing Na(+) with an affinity that is fivefold higher than for G-quadruplex structures formed in the presence of K(+). However, the binding of the second heterodimer is insensitive to DNA secondary structure, presumably due to unfolding resulting from binding of the first POT1-TPP1. We further show that the rate constant for POT1-TPP1-induced unfolding of DNA secondary structure is substantially faster for G-quadruplex topologies formed in the presence of Na(+) ions. When bound to DNA, POT1-TPP1 forms complexes with similar CD spectra and enhances telomerase activity for all DNA substrates tested, regardless of the substrate secondary structure or solution monovalent ion composition. Together, these data indicate that binding of POT1-TPP1 unfolds telomere secondary structure to assist loading of additional heterodimers and to ensure efficient promotion of telomerase-mediated extension.

Indexed as

AminopeptidasesDipeptidyl-Peptidases and Tripeptidyl-PeptidasesDNA, Single-StrandedG-QuadruplexesHumansPolymorphism, GeneticProtein BindingSerine ProteasesShelterin ComplexTelomeraseTelomereTelomere-Binding ProteinsACD protein, humanAminopeptidasesDipeptidyl-Peptidases and Tripeptidyl-PeptidasesDNA, Single-StrandedPOT1 protein, humanSerine ProteasesShelterin ComplexTelomeraseTelomere-Binding ProteinsG-quadruplexshelterintelomerasetelomere

Identifiers

PMID27173378
PMCPMC4905795

What OpenQuestion holds

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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.