Evidence map›Paper›PMID 27168798›Full record

ArticleExperimental and therapeutic medicine2016

NSAIDs modulate GABA-activated currents via Ca

Lei Zhao, L I Li, Ke-Tao Ma, Yang Wang, Jing Li, Wen-Yan Shi, H E Zhu, Zhong-Shuang Zhang, Jun-Qiang Si

Open access · diamondAbstract read
PubMed Full text at PMC Publisher
In one paragraph

Article in Experimental and therapeutic medicine, 2016. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 6 papers.

0numbers the graph read from it
0cells of the map it votes in
6citing papers in PubMed
1.0field-weighted citation impact, top 22% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

6 citing papers in PubMed, 10 citations in OpenAlex.

  1. Article
  2. Nociceptor Signalling through ion Channel Regulation via GPCRs.International journal of molecular sciences · 2019
    Review
  3. Article
  4. TMEM16A and TMEM16B channel proteins generate CaThe Journal of biological chemistry · 2018
    Article
  5. Article
  6. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors at 1 institution in 1 country.

Lei ZhaoDepartment of Physiology, Shihezi University Medical College, Shihezi, Xinjiang 832002, P.R. China; Electrophysiological Laboratory, Laboratory of Xinjiang Endemic and Ethnic Diseases, Shihezi University Medical College, Shihezi, Xinjiang 832002, P.R. China.
L I LiDepartment of Physiology, Shihezi University Medical College, Shihezi, Xinjiang 832002, P.R. China; Electrophysiological Laboratory, Laboratory of Xinjiang Endemic and Ethnic Diseases, Shihezi University Medical College, Shihezi, Xinjiang 832002, P.R. China.
Ke-Tao MaDepartment of Physiology, Shihezi University Medical College, Shihezi, Xinjiang 832002, P.R. China; Electrophysiological Laboratory, Laboratory of Xinjiang Endemic and Ethnic Diseases, Shihezi University Medical College, Shihezi, Xinjiang 832002, P.R. China.
Yang WangDepartment of Physiology, Shihezi University Medical College, Shihezi, Xinjiang 832002, P.R. China; Electrophysiological Laboratory, Laboratory of Xinjiang Endemic and Ethnic Diseases, Shihezi University Medical College, Shihezi, Xinjiang 832002, P.R. China; Department of Physiology, School of Basic Medical Sciences, Wuhan University, Wuhan, Hubei 430071, P.R. China.
Jing LiDepartment of Physiology, Shihezi University Medical College, Shihezi, Xinjiang 832002, P.R. China.
Wen-Yan ShiDepartment of Physiology, Shihezi University Medical College, Shihezi, Xinjiang 832002, P.R. China; Electrophysiological Laboratory, Laboratory of Xinjiang Endemic and Ethnic Diseases, Shihezi University Medical College, Shihezi, Xinjiang 832002, P.R. China.
H E ZhuDepartment of Physiology, Shihezi University Medical College, Shihezi, Xinjiang 832002, P.R. China; Electrophysiological Laboratory, Laboratory of Xinjiang Endemic and Ethnic Diseases, Shihezi University Medical College, Shihezi, Xinjiang 832002, P.R. China.
Zhong-Shuang ZhangDepartment of Physiology, Shihezi University Medical College, Shihezi, Xinjiang 832002, P.R. China; Electrophysiological Laboratory, Laboratory of Xinjiang Endemic and Ethnic Diseases, Shihezi University Medical College, Shihezi, Xinjiang 832002, P.R. China.
Jun-Qiang SiDepartment of Physiology, Shihezi University Medical College, Shihezi, Xinjiang 832002, P.R. China; Electrophysiological Laboratory, Laboratory of Xinjiang Endemic and Ethnic Diseases, Shihezi University Medical College, Shihezi, Xinjiang 832002, P.R. China; Department of Physiology, School of Basic Medical Sciences, Wuhan University, Wuhan, Hubei 430071, P.R. China; Department of Physiology, School of Basic Medical Sciences, Huazhong University of Science and Technology, Wuhan, Hubei 430030, P.R. China.
Shihezi University · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The ability of non-steroidal anti-inflammatory drugs (NSAIDs) to modulate γ-aminobutyrate (GABA)-activated currents via Ca

Indexed as

Ca2+-activated Cl− channelsdorsal root ganglionmodulatoryniflumic acidnon-steroidal anti-inflammatory drugsγ-aminobutyrate-induced inward current

Identifiers

PMID27168798
PMCPMC4840517
OpenAlexW2298739593

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.