ReviewAntibiotics (Basel, Switzerland)2013
Multiple Pathways of Genome Plasticity Leading to Development of Antibiotic Resistance.
Review in Antibiotics (Basel, Switzerland), 2013. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 17 papers, 1 of them a synthesis that pooled it.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
17 citing papers in PubMed, 1 synthesis or guideline pooled it.
- Pooled it
- tRNA-modifying enzymes in bacterial stress adaptation.Open biology · 2025Review
- Antimicrobial Residues in Poultry Litter: Assessing the Association of Antimicrobial Persistence with ResistantAntibiotics (Basel, Switzerland) · 2025Article
- A one health approach for monitoring antimicrobial resistance: developing a national freshwater pilot effort.Frontiers in water · 2024Article
- Exploring Antimicrobial Resistance Profiles ofMicroorganisms · 2023Article
- Resistance to Critical Important Antibacterials inAntibiotics (Basel, Switzerland) · 2022Article
- Endophytic Fungi as a Source of Antibacterial Compounds-A Focus on Gram-Negative Bacteria.Antibiotics (Basel, Switzerland) · 2022Review
- Effects of Sub-Minimum Inhibitory Concentrations of Imipenem and Colistin on Expression of Biofilm-Specific Antibiotic Resistance and Virulence Genes inInternational journal of molecular sciences · 2022Article
- HAM-ART: An optimised culture-free Hi-C metagenomics pipeline for tracking antimicrobial resistance genes in complex microbial communities.PLoS genetics · 2022Article
- Subinhibitory antibiotic concentrations promote the horizontal transfer of plasmid-borne resistance genes fromFrontiers in microbiology · 2022Article
- Life Within a Contaminated Niche: Comparative Genomic Analyses of an Integrative Conjugative Element ICEFrontiers in microbiology · 2022Article
- Antibiotic Resistance - A Cause for Reemergence of Infections.Indian journal of pediatrics · 2020Review
- Ciprofloxacin, amoxicillin, and aminoglycosides stimulate genetic and phenotypic changes in uropathogenic Escherichia coli strains.Virulence · 2019Article
- Article
- Constraints on lateral gene transfer in promoting fimbrial usher protein diversity and function.Open biology · 2017Article
- The Current Case of Quinolones: Synthetic Approaches and Antibacterial Activity.Molecules (Basel, Switzerland) · 2016Review
- The antimicrobial resistance crisis: causes, consequences, and management.Frontiers in public health · 2014Review
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
3 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
The emergence of multi-resistant bacterial strains is a major source of concern and has been correlated with the widespread use of antibiotics. The origins of resistance are intensively studied and many mechanisms involved in resistance have been identified, such as exogenous gene acquisition by horizontal gene transfer (HGT), mutations in the targeted functions, and more recently, antibiotic tolerance through persistence. In this review, we focus on factors leading to integron rearrangements and gene capture facilitating antibiotic resistance acquisition, maintenance and spread. The role of stress responses, such as the SOS response, is discussed.
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.