Evidence map›Paper›PMID 26950180›Full record

ArticleMolecular plant pathology2017

The putative phospholipase Lip2 counteracts oxidative damage and influences the virulence of Ustilago maydis.

Scott C Lambie, Matthias Kretschmer, Daniel Croll, Tegan M Haslam, Ljerka Kunst, Jana Klose, James W Kronstad

Open access · bronzeAbstract read
In one paragraph

Article in Molecular plant pathology, 2017. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.

0numbers the graph read from it
0cells of the map it votes in
4citing papers in PubMed
–field-weighted citation impact, top 99% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

4 citing papers in PubMed, 7 citations in OpenAlex.

  1. Article
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  3. Review
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors at 1 institution in 1 country.

Scott C LambieDepartment of Microbiology and Immunology, University of British Columbia, Vancouver V6T 1Z3, BC, Canada.
Matthias KretschmerThe Michael Smith Laboratories, University of British Columbia, Vancouver V6T 1Z4, BC, Canada.
Daniel CrollThe Michael Smith Laboratories, University of British Columbia, Vancouver V6T 1Z4, BC, Canada.
Tegan M HaslamDepartment of Botany, University of British Columbia, Vancouver V6T 1Z4, BC, Canada.
Ljerka KunstDepartment of Botany, University of British Columbia, Vancouver V6T 1Z4, BC, Canada.
Jana KloseDepartment of Microbiology and Immunology, University of British Columbia, Vancouver V6T 1Z3, BC, Canada.
James W KronstadDepartment of Microbiology and Immunology, University of British Columbia, Vancouver V6T 1Z3, BC, Canada.
University of British Columbia · CA

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Ustilago maydis is an obligate biotrophic fungal pathogen which causes common smut disease of corn. To proliferate in host tissue, U. maydis must gain access to nutrients and overcome plant defence responses, such as the production of reactive oxygen species. The elucidation of the mechanisms by which U. maydis meets these challenges is critical for the development of strategies to combat smut disease. In this study, we focused on the contributions of phospholipases (PLs) to the pathogenesis of corn smut disease. We identified 11 genes encoding putative PLs and characterized the transcript levels for these genes in the fungus grown in culture and during infection of corn tissue. To assess the contributions of specific PLs, we focused on two genes, lip1 and lip2, which encode putative phospholipase A

Indexed as

Oxidative StressCardiolipinsCell RespirationFungal ProteinsGene DeletionGenome, FungalHaploidyMutationPhospholipasesRNA, MessengerUstilagoVirulenceCardiolipinsFungal ProteinsPhospholipasesRNA, Messengercornfungal pathogenesismaizemitochondriasmut disease

Identifiers

PMID26950180
PMCPMC6638309
OpenAlexW2296548066

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.