Evidence map›Paper›PMID 26932189›Full record

ArticleEuropean journal of human genetics : EJHG2016

A polymorphic Alu insertion that mediates distinct disease-associated deletions.

Amir Jahic, Anne K Erichsen, Thomas Deufel, Chantal M Tallaksen, Christian Beetz

Abstract read
In one paragraph

Article in European journal of human genetics : EJHG, 2016. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.

0numbers the graph read from it
0cells of the map it votes in
4citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

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Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

4 citing papers in PubMed.

  1. International journal of molecular sciences · 2024
    Article
  2. Article
  3. AnNAR genomics and bioinformatics · 2022
    Article
  4. RNA Editing and Retrotransposons in Neurology.Frontiers in molecular neuroscience · 2018
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors.

Amir JahicDepartment of Clinical Chemistry and Laboratory Medicine, Jena University Hospital, Jena, Germany.
Anne K ErichsenDepartment of Neurology, Oslo University Hospital, Oslo, Norway.
Thomas DeufelDepartment of Clinical Chemistry and Laboratory Medicine, Jena University Hospital, Jena, Germany.ORCID 0000-0003-4839-3117
Chantal M TallaksenDepartment of Neurology, Oslo University Hospital, Oslo, Norway.
Christian BeetzDepartment of Clinical Chemistry and Laboratory Medicine, Jena University Hospital, Jena, Germany.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Large deletions that are associated with insertions of Alu-derived sequence represent a rare, but potentially unique class of alterations. Whether they form by a one-step mechanism or by a primary insertion step followed by an independent secondary deletion step is not clear. We resolved two disease-associated SPAST deletions, which involve distinct exons by long range PCR. Alu-derived sequence was observed between the breakpoints in both cases. The intronic regions that represent the targets of potentially involved Alu retrotransposition events overlapped. Microsatellite- and SNP-based haplotyping indicated that both deletions originated on one and the same founder allele. Our data suggest that the deletions are best explained by two-step insertion-deletion scenarios for which a single Alu retrotransposition event represents the shared primary step. This Alu then mediated one of the deletions by non-homologous end joining and the other by non-allelic homologous recombination. Our findings thus strongly argue for temporal separation of insertion and deletion in Alu insertion-associated deletions. They also suggest that certain Alu integrations confer a general increase in local genomic instability, and that this explains why they are usually not detected during the probably short time that precedes the rearrangements they mediate.

Indexed as

Mutagenesis, InsertionalPolymorphism, GeneticAdenosine TriphosphatasesAllelesAlu ElementsChromosome BreakpointsExonsGene DeletionHomologous RecombinationHumansParaplegiaSpastic Paraplegia, HereditarySpastinAdenosine TriphosphatasesSpastinSPAST protein, human

Identifiers

PMID26932189
PMCPMC4989209

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.