ArticleMolecular plant pathology2017
Function of the genetic element 'Mona' associated with fungicide resistance in Monilinia fructicola.
Article in Molecular plant pathology, 2017. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 11 papers.
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Who cites it
11 citing papers in PubMed.
- The MoSR-MoDde1 Regulatory Axis: A Novel Determinant of DMI Sensitivity and a Target for Sustainable Rice Blast Control.Advanced science (Weinheim, Baden-Wurttemberg, Germany) · 2026Article
- Involvement ofJournal of fungi (Basel, Switzerland) · 2026Article
- The poplar pathogenMicrobial genomics · 2026Article
- Involvement of the SIX10 Gene in the Pathogenicity ofInternational journal of molecular sciences · 2025Article
- Article
- Identification of propranolol and derivatives that are chemical inhibitors of phosphatidate phosphatase as potential broad-spectrum fungicides.Plant communications · 2024Article
- Article
- Article
- MfOfd1 is crucial for stress responses and virulence in the peach brown rot fungus Monilinia fructicola.Molecular plant pathology · 2020Article
- Labeling ofGenes · 2019Article
- A Putative ZnFrontiers in microbiology · 2018Article
Corrections and comments
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Authors and funding
4 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
The genetic element 'Mona' has been shown previously to be associated with resistance to demethylation inhibitors (DMIs) in Monilinia fructicola. In this study, the promoter activity of the 'Mona' element was demonstrated genetically and the activity was narrowed down to a 20-bp active region through a series of deletions. 'Mona' knockout transformants (ΔMona) were generated from DMI-resistant isolate Bmpc7, and EC
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