Evidence map›Paper›PMID 26853723›Full record

ArticleCellular & molecular immunology2017

Trans-presentation of interleukin-15 by interleukin-15 receptor alpha is dispensable for the pathogenesis of autoimmune type 1 diabetes.

Diwakar Bobbala, Marian Mayhue, Alfredo Menendez, Subburaj Ilangumaran, Sheela Ramanathan

Open access · bronzeAbstract read
In one paragraph

Article in Cellular & molecular immunology, 2017. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 12 papers.

0numbers the graph read from it
0cells of the map it votes in
12citing papers in PubMed
0.8field-weighted citation impact, top 29% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

12 citing papers in PubMed, 23 citations in OpenAlex.

  1. Article
  2. Interleukin 15 and autoimmune disorders: pathophysiology, therapeutic potential, and clinical implications.Inflammation research : official journal of the European Histamine Research Society ... [et al.] · 2025
    Review
  3. Article
  4. Mapping novel QTL and fine mapping of previously identified QTL associated with glucose tolerance using the collaborative cross mice.Mammalian genome : official journal of the International Mammalian Genome Society · 2024
    Article
  5. Article
  6. Article
  7. Antigen-presenting B cells promote TCF-1Frontiers in immunology · 2023
    Article
  8. Article
  9. Article
  10. Article
  11. Review
  12. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 1 institution in 1 country.

Diwakar BobbalaImmunology Division, Department of Pediatrics, Université de Sherbrooke, Sherbrooke, QC, Canada.
Marian MayhueImmunology Division, Department of Pediatrics, Université de Sherbrooke, Sherbrooke, QC, Canada.
Alfredo MenendezDepartment of Microbiology, Faculty of Medicine and Health Sciences, Université de Sherbrooke, Sherbrooke, QC, Canada.
Subburaj IlangumaranImmunology Division, Department of Pediatrics, Université de Sherbrooke, Sherbrooke, QC, Canada.
Sheela RamanathanImmunology Division, Department of Pediatrics, Université de Sherbrooke, Sherbrooke, QC, Canada.
Université de Sherbrooke · CA

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Interleukin-15 (IL-15) is a pro-inflammatory cytokine that is required for the survival and activation of memory CD8

Indexed as

AnimalsAntigens, ViralCD8-Positive T-LymphocytesCross-PrimingDiabetes Mellitus, Type 1Interleukin-15Interleukin-15 Receptor alpha SubunitLymphocytic ChoriomeningitisLymphocytic choriomeningitis virusMice, Inbred C57BLMice, Inbred NODRatsAntigens, ViralInterleukin-15Interleukin-15 Receptor alpha Subunit

Identifiers

PMID26853723
PMCPMC5520411
OpenAlexW2266627318

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.