Evidence map›Paper›PMID 26839113›Full record

ReviewHuman genetics2016

Improved prediction of complex diseases by common genetic markers: state of the art and further perspectives.

Bent Müller, Arndt Wilcke, Anne-Laure Boulesteix, Jens Brauer, Eberhard Passarge, Johannes Boltze, Holger Kirsten

Abstract readReview
In one paragraph

Review in Human genetics, 2016. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 19 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
19citing papers in PubMed, 1 pooled it
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

19 citing papers in PubMed, 1 synthesis or guideline pooled it.

  1. Pooled it
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  6. Article
  7. Psychosomatic Aspects of The Development of Comorbid Pathology: A Review.Medical journal of the Islamic Republic of Iran · 2022
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  15. Brain and behavior · 2017
    Article
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors.

Bent MüllerDepartment of Cell Therapy, Fraunhofer Institute for Cell Therapy and Immunology, Perlickstraße 1, Leipzig, Germany. bent.mueller@izi.fraunhofer.de.
Arndt WilckeDepartment of Cell Therapy, Fraunhofer Institute for Cell Therapy and Immunology, Perlickstraße 1, Leipzig, Germany.
Anne-Laure BoulesteixDepartment of Medical Informatics, Biometry and Epidemiology, University of Munich, Munich, Germany.
Jens BrauerDepartment of Neuropsychology, Max Planck Institute for Human Cognitive and Brain Sciences, Leipzig, Germany.
Eberhard PassargeInstitute for Human Genetics, University Hospital Leipzig, University of Leipzig, Leipzig, Germany.
Johannes BoltzeDepartment of Cell Therapy, Fraunhofer Institute for Cell Therapy and Immunology, Perlickstraße 1, Leipzig, Germany.
Holger KirstenDepartment of Cell Therapy, Fraunhofer Institute for Cell Therapy and Immunology, Perlickstraße 1, Leipzig, Germany.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Reliable risk assessment of frequent, but treatable diseases and disorders has considerable clinical and socio-economic relevance. However, as these conditions usually originate from a complex interplay between genetic and environmental factors, precise prediction remains a considerable challenge. The current progress in genotyping technology has resulted in a substantial increase of knowledge regarding the genetic basis of such diseases and disorders. Consequently, common genetic risk variants are increasingly being included in epidemiological models to improve risk prediction. This work reviews recent high-quality publications targeting the prediction of common complex diseases. To be included in this review, articles had to report both, numerical measures of prediction performance based on traditional (non-genetic) risk factors, as well as measures of prediction performance when adding common genetic variants to the model. Systematic PubMed-based search finally identified 55 eligible studies. These studies were compared with respect to the chosen approach and methodology as well as results and clinical impact. Phenotypes analysed included tumours, diabetes mellitus, and cardiovascular diseases. All studies applied one or more statistical measures reporting on calibration, discrimination, or reclassification to quantify the benefit of including SNPs, but differed substantially regarding the methodological details that were reported. Several examples for improved risk assessments by considering disease-related SNPs were identified. Although the add-on benefit of including SNP genotyping data was mostly moderate, the strategy can be of clinical relevance and may, when being paralleled by an even deeper understanding of disease-related genetics, further explain the development of enhanced predictive and diagnostic strategies for complex diseases.

Indexed as

Genetic MarkersCardiovascular DiseasesDiabetes MellitusGenotyping TechniquesHumansNeoplasmsPolymorphism, Single NucleotideRisk AssessmentGenetic Markers

Identifiers

PMID26839113
PMCPMC4759222

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.