Evidence map›Paper›PMID 26803516›Full record

ArticleTumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine2016

ING3 is associated with increased cell invasion and lethal outcome in ERG-negative prostate cancer patients.

Amal Almami, Samar A Hegazy, Arash Nabbi, Mohammed Alshalalfa, Asma Salman, Hatem Abou-Ouf, Karl Riabowol, Tarek A Bismar

Abstract read
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In one paragraph

Article in Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine, 2016. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 9 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
9citing papers in PubMed, 1 pooled it
1.0field-weighted citation impact, top 22% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

9 citing papers in PubMed, 1 synthesis or guideline pooled it, 15 citations in OpenAlex.

  1. Pooled it
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors at 3 institutions in 1 country.

Amal AlmamiMedical Science Department Faculty of Medicine, University of Calgary, Calgary, Alberta, Canada.
Samar A HegazyDepartment of Pathology and Laboratory Medicine, University of Calgary and Calgary Laboratory Services, Calgary, Alberta, Canada.
Arash NabbiMedical Science Department Faculty of Medicine, University of Calgary, Calgary, Alberta, Canada.
Mohammed AlshalalfaThe Prostate Centre, Calgary, Alberta, Canada.
Asma SalmanDepartment of Pathology and Laboratory Medicine, University of Calgary and Calgary Laboratory Services, Calgary, Alberta, Canada.
Hatem Abou-OufDepartment of Pathology and Laboratory Medicine, University of Calgary and Calgary Laboratory Services, Calgary, Alberta, Canada.
Karl RiabowolDepartment of Pathology and Laboratory Medicine, University of Calgary and Calgary Laboratory Services, Calgary, Alberta, Canada.
Tarek A BismarDepartment of Pathology and Laboratory Medicine, University of Calgary and Calgary Laboratory Services, Calgary, Alberta, Canada. tarek.bismar@cls.ab.ca.
University of Calgary · CACalgary Laboratory Services · CAProstate Cancer Canada · CA

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The inhibitor of growth family member 3 (ING3) is a member of the ING tumor suppressor family. Although its expression has been reported in various types of cancers, the role of ING3 and its prognostic value in prostate cancer (PCa) has not been investigated. ING3 expression and prognostic value was assessed in a cohort of PCa patients (n = 312) treated with transurethral resection of prostate using immumoflourescent automated quantitative analysis (AQUA) system. In vitro studies were carried out in conjunction to investigate its expression in various PCa cell lines. ING3 knockdown was also carried out in DU145 cell lines to assess for any changes in invasion and migration. ING3 expression was highest in benign prostate tissues (mean 3.2 ± 0.54) compared to PCa (mean 2.5 ± 0.26) (p = 0.437), advanced prostate cancer (AdvPCa) (mean 1.5 ± 0.32) (p = 0.004), and castration-resistant prostate cancer (CRPC) (mean 2.28 ± 0.32) (p = 0.285). ING3 expression was inversely correlated to Gleason score (p = 0.039) and ETS-related gene (ERG) expression (p = 0.019). Higher ING3 expression was marginally associated with lethal disease (p = 0.052), and this was more pronounced in patients with ERG-negative status (p = 0.018). Inhibition of ING3 in DU145 PCa cells using small interfering RNA (siRNA) was associated with decreased cell invasion (p = 0.0016) and cell migration compared to control cells. ING3 is significantly associated with PCa disease progression and cancer-specific mortality. To our knowledge, this is the first report suggesting an oncogenic function of ING3, previously well known as a tumor suppressor protein. Further studies should investigate potential-related pathways in association to ING3.

Indexed as

Cell MovementApoptosisBiomarkers, TumorBlotting, WesternCell ProliferationCohort StudiesDisease ProgressionFollow-Up StudiesHomeodomain ProteinsHumansImmunoenzyme TechniquesMaleNeoplasm GradingNeoplasm InvasivenessNeoplasm StagingPrognosisAR protein, humanBiomarkers, TumorERG protein, humanHomeodomain ProteinsING3 protein, humanReceptors, AndrogenRNA, MessengerTranscriptional Regulator ERGTumor Suppressor ProteinsING3InvasionMetastasisOncogeneProgressionProstate cancerTumor suppressor

Identifiers

PMID26803516
OpenAlexW2296430625

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.