A review of the literature on cardiac electrical activity between fibroblasts and myocytes.

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

Who cites it

17 citing papers in PubMed.

1. Interstitial cells and arrhythmia.American journal of physiology. Cell physiology · 2026
   Review
2. Gap junctional and ephaptic coupling in cardiac electrical propagation: homocellular and heterocellular perspectives.The Journal of physiology · 2026
   Review
3. Exploring the Impact of BKInternational journal of molecular sciences · 2024
   Review
4. Fibroblasts in heart scar tissue directly regulate cardiac excitability and arrhythmogenesis.Science (New York, N.Y.) · 2023
   Article
5. Progress on role of ion channels of cardiac fibroblasts in fibrosis.Frontiers in physiology · 2023
   Review
6. Carbon monoxide activates large-conductance calcium-activated potassium channels of human cardiac fibroblasts through various mechanisms.The Korean journal of physiology & pharmacology : official journal of the Korean Physiological Society and the Korean Society of Pharmacology · 2021
   Article
7. Connexin43 expression in bone marrow derived cells contributes to the electrophysiological properties of cardiac scar tissue.Scientific reports · 2020
   Article
8. Cardiomyocyte-myofibroblast contact dynamism is modulated by connexin-43.FASEB journal : official publication of the Federation of American Societies for Experimental Biology · 2019
   Article
9. Strength-duration relationship as a tool to prioritize cardiac tissue properties that govern electrical excitability.American journal of physiology. Heart and circulatory physiology · 2019
   Article
10. Reduced cardiomyocyte NaJournal of cellular physiology · 2019
    Article
11. Personalized Cardiac Computational Models: From Clinical Data to Simulation of Infarct-Related Ventricular Tachycardia.Frontiers in physiology · 2019
    Article
12. Ventricular pro-arrhythmic phenotype, arrhythmic substrate, ageing and mitochondrial dysfunction in peroxisome proliferator activated receptor-γ coactivator-1β deficient (Pgc-1βMechanisms of ageing and development · 2018
    Article
13. Effects of Nitric Oxide on Voltage-Gated K⁺ Currents in Human Cardiac Fibroblasts through the Protein Kinase G and Protein Kinase A Pathways but Not through S-Nitrosylation.International journal of molecular sciences · 2018
    Article
14. Physiologic, Pathologic, and Therapeutic Paracrine Modulation of Cardiac Excitation-Contraction Coupling.Circulation research · 2018
    Review
15. Directed fusion of cardiac spheroids into larger heterocellular microtissues enables investigation of cardiac action potential propagation via cardiac fibroblasts.PloS one · 2018
    Article
16. GAmerican journal of physiology. Heart and circulatory physiology · 2017
    Article
17. Electrotonic coupling of excitable and nonexcitable cells in the heart revealed by optogenetics.Proceedings of the National Academy of Sciences of the United States of America · 2016
    Article

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

Authors and funding

3 authors.

Funding

Abstract

Indexed as