Evidence map›Paper›PMID 26710305›Full record

ReviewClinical obstetrics and gynecology2016

Fibroids: Genotype and Phenotype.

Zehra Ordulu

Abstract readReview
In one paragraph

Review in Clinical obstetrics and gynecology, 2016. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 7 papers.

0numbers the graph read from it
0cells of the map it votes in
7citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

7 citing papers in PubMed.

  1. Article
  2. Article
  3. Article
  4. Review
  5. Molecular and clinicopathologic characterization of intravenous leiomyomatosis.Modern pathology : an official journal of the United States and Canadian Academy of Pathology, Inc · 2020
    Article
  6. Article
  7. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

1 author.

Zehra Ordulu*Department of Obstetrics, Gynecology and Reproductive Biology, Brigham and Women's Hospital †Department of Pathology, Massachusetts General Hospital ‡Harvard Medical School, Boston, Massachusetts.

Funding

Genetic Studies of Uterine LeiomyomataR01HD060530 · NICHD · BRIGHAM AND WOMEN'S HOSPITAL · PI MORTON, CYNTHIA CASSON · 2010 to 2015
$1.8M
NICHD NIH HHS R01 HD060530NICHD NIH HHS R01HD060530
6 · The paper itself

Abstract

Fibroids represent a major public health care problem as the most prevalent pelvic tumors in women of reproductive age and as the leading cause of gynecologic surgeries in the United States. The recent advances in the genomic technologies including genome-wide association studies and high-throughput sequencing provide insight into their pathogenesis and molecular classification. Understanding the molecular basis of fibroids may facilitate development of effective targeted treatment options of this very common disease.

Indexed as

Collagen Type IVDNA-Binding ProteinsEpigenesis, GeneticFatty Acid Synthase, Type IFemaleGene DeletionGenetic VariationGenome-Wide Association StudyGenotypeHistone AcetyltransferasesHMGA ProteinsHomeodomain ProteinsHumansLeiomyomaMediator ComplexMutationCOL4A5 protein, humanCOL4A6 protein, humanCollagen Type IVCUX1 protein, humanDNA-Binding ProteinsFASN protein, humanFatty Acid Synthase, Type IHistone AcetyltransferasesHMGA ProteinsHomeodomain ProteinsKAT6B protein, humanMED12 protein, humanMediator ComplexNuclear ProteinsRAD51B protein, humanRepressor ProteinsTranscription Factors

Identifiers

PMID26710305
PMCPMC4733577

What OpenQuestion holds

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Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.