Evidence map›Paper›PMID 26612463›Full record

ArticleModern pathology : an official journal of the United States and Canadian Academy of Pathology, Inc2016

Integrated analysis of the genomic instability of PTEN in clinically insignificant and significant prostate cancer.

Stephen J Murphy, Robert J Karnes, Farhad Kosari, B Edgardo R Parilla Castellar, Benjamin R Kipp, Sarah H Johnson, Simone Terra, Faye R Harris, Geoffrey C Halling, Janet L Schaefer Klein and 6 more

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Article in Modern pathology : an official journal of the United States and Canadian Academy of Pathology, Inc, 2016. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 20 papers.

0numbers the graph read from it
0cells of the map it votes in
20citing papers in PubMed
5.0field-weighted citation impact, top 4% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

20 citing papers in PubMed, 47 citations in OpenAlex.

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  15. Distinct subtypes of genomicMolecular cytogenetics · 2018
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

16 authors at 3 institutions in 1 country.

Stephen J MurphyBiomarker Discovery Program, Center of Individualized Medicine, Mayo Clinic, Rochester, MN, USA.
Robert J KarnesDepartment of Urology, Mayo Clinic, Rochester, MN, USA.
Farhad KosariBiomarker Discovery Program, Center of Individualized Medicine, Mayo Clinic, Rochester, MN, USA.
B Edgardo R Parilla CastellarDepartment of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN, USA.
Benjamin R KippDepartment of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN, USA.
Sarah H JohnsonBiomarker Discovery Program, Center of Individualized Medicine, Mayo Clinic, Rochester, MN, USA.
Simone TerraBiomarker Discovery Program, Center of Individualized Medicine, Mayo Clinic, Rochester, MN, USA.
Faye R HarrisBiomarker Discovery Program, Center of Individualized Medicine, Mayo Clinic, Rochester, MN, USA.
Geoffrey C HallingBiomarker Discovery Program, Center of Individualized Medicine, Mayo Clinic, Rochester, MN, USA.
Janet L Schaefer KleinBiomarker Discovery Program, Center of Individualized Medicine, Mayo Clinic, Rochester, MN, USA.
Aqsa NasirBiomarker Discovery Program, Center of Individualized Medicine, Mayo Clinic, Rochester, MN, USA.
Eric BergstrahlDepartment of Health Sciences Research, Mayo Clinic, Rochester, MN, USA.
Laureano J RangelDepartment of Health Sciences Research, Mayo Clinic, Rochester, MN, USA.
William R SukovDepartment of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN, USA.
George VasmatzisBiomarker Discovery Program, Center of Individualized Medicine, Mayo Clinic, Rochester, MN, USA.
John C ChevilleBiomarker Discovery Program, Center of Individualized Medicine, Mayo Clinic, Rochester, MN, USA.
Mayo Clinic in Arizona · USMayo Clinic · USMayo Clinic in Florida · US

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Patients with clinically insignificant prostate cancer remain a major over-treated population. PTEN loss is one of the most recurrent alterations in prostate cancer associated with an aggressive phenotype, however, the occurrence of PTEN loss in insignificant prostate cancer has not been reported and its role in the separation of insignificant from significant prostate cancer is unclear. An integrated analysis of PTEN loss was, therefore, performed for structural variations, point mutations and protein expression in clinically insignificant (48 cases) and significant (76 cases) prostate cancers treated by radical prostatectomy. Whole-genome mate pair sequencing was performed on tumor cells isolated by laser capture microdissection to characterize PTEN structural alterations. Fluorescence in situ hybridization probes were constructed from the sequencing data to detect the spectrum of these PTEN alterations. PTEN loss by mate pair sequencing and fluorescence in situ hybridization occurred in 2% of insignificant, 13% of large volume Gleason score 6, and 46% of Gleason score 7 and higher cancers. In Gleason score 7 cancers with PTEN loss, PTEN alterations were detected in both Gleason pattern 3 and 4 in 57% of cases by mate pair sequencing, 75% by in situ hybridization and 86% by immunohistochemistry. PTEN loss by sequencing was strongly associated with TMPRSS2-ERG fusion, biochemical recurrence, PTEN loss by in situ hybridization and protein loss by immunohistochemistry. The complex nature of PTEN rearrangements was unveiled by sequencing, detailing the heterogeneous events leading to homozygous loss of PTEN. PTEN point mutation was present in 5% of clinically significant tumors and not in insignificant cancer or high-grade prostatic intraepithelial neoplasia. PTEN loss is infrequent in clinically insignificant prostate cancer, and is associated with higher grade tumors. Detection of PTEN loss in Gleason score 6 cancer in a needle biopsy specimen indicates a higher likelihood of clinically significant prostate cancer.

Indexed as

Genomic InstabilityAdultAgedAged, 80 and overBiomarkers, TumorBiopsy, NeedleDNA Mutational AnalysisGene FusionGene RearrangementGenetic Predisposition to DiseaseHumansImmunohistochemistryIn Situ Hybridization, FluorescenceMaleMiddle AgedNeoplasm GradingBiomarkers, TumorOncogene Proteins, FusionPTEN PhosphohydrolasePTEN protein, humanTMPRSS2-ERG fusion protein, human

Identifiers

PMID26612463
OpenAlexW2181108066

What OpenQuestion holds

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Read underepoch 390

Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.