ReviewZeitschrift fur Rheumatologie2015
[Hypertrophic chondrocytes: Programmed cell death or stem cell reservoir?].
Review in Zeitschrift fur Rheumatologie, 2015. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
1 citing paper in PubMed.
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
2 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
The majority of bones in the vertebrate skeleton develop by endochondral ossification, a process during which an intermediate cartilage template is successively replaced by bone. Many aspects of this process are relatively well understood; nevertheless, the origin of trabecular bone-forming osteoblasts and mesenchymal stem cells of the stroma has long remained under debate. Until recently, progenitors of these cell types were thought to enter the bone-forming structures from the periosteum together with the invading vasculature. Recent unexpected results revealed, however, that under physiological conditions differentiated hypertrophic chondrocytes give rise to both, osteoblasts and mesenchymal progenitor cells, thereby contributing to the formation of trabecular bone and bone marrow.
Indexed as
Identifiers
26555549What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.