ArticleDevelopment (Cambridge, England)2015
A transport and retention mechanism for the sustained distal localization of Spn-F-IKKε during Drosophila bristle elongation.
Article in Development (Cambridge, England), 2015. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
2 citing papers in PubMed.
- Ik2/TBK1 and Hook/Dynein, an adaptor complex for early endosome transport, are genetic modifiers of FTD-associated mutant CHMP2B toxicity in Drosophila.Scientific reports · 2020Article
- GFP-Forked, a genetic reporter for studyingBiology open · 2019Article
Corrections and comments
- Erratum for
Authors and funding
6 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
There was an error published in Development 142, 2338-2351. Otani et al. reported the genetic interactions between ikkε and spn-F, using the allele ikkε66. This allele was referred to in the Materials and Methods on p. 2349, Fig. 3 on p. 2343 and Table S1. However, they subsequently found that the allele used in the experiments was ikkε1 (also known as ikkε36). This was as a result of misannotation in their laboratory stock list. Both alleles are strong loss-of-function alleles with a missense mutation in the kinase domain and show similar phenotypes (Oshima et al., 2006; Shapiro and Anderson, 2006). Therefore, this error does not affect the conclusions of the paper. The authors apologise to readers for this mistake.
Identifiers
What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.