ArticleIndoor air2016
Chronic exposure to biomass fuel smoke and markers of endothelial inflammation.
Article in Indoor air, 2016. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 17 papers.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Who cites it
17 citing papers in PubMed, 32 citations in OpenAlex.
- From Exposure to Outcome: Air Pollution-Induced Oxidative Stress as a Determinant of Early and Late Outcomes After Coronary Artery Bypass Grafting.Antioxidants (Basel, Switzerland) · 2026Review
- Do Circulating Extracellular Vesicles Strictly Reflect Bronchoalveolar Lavage Extracellular Vesicles in COPD?International journal of molecular sciences · 2023Article
- Household air pollution from wood-burning cookstoves and C-reactive protein among women in rural Honduras.International journal of hygiene and environmental health · 2022Article
- The Effect of Chronic Inhouse Biomass Fuel Smoke Exposure on Coronary Slow Flow Phenomenon in Women Living in the Eastern Region of Turkey.Medeniyet medical journal · 2022Article
- Household air pollution and blood markers of inflammation: A cross-sectional analysis.Indoor air · 2021Article
- Exposure to household air pollutants and endothelial dysfunction in rural Bangladesh: A cross-sectional study.Environmental epidemiology (Philadelphia, Pa.) · 2021Article
- Indoor air pollution concentrations and cardiometabolic health across four diverse settings in Peru: a cross-sectional study.Environmental health : a global access science source · 2020Article
- Design and Rationale of the HAPIN Study: A Multicountry Randomized Controlled Trial to Assess the Effect of Liquefied Petroleum Gas Stove and Continuous Fuel Distribution.Environmental health perspectives · 2020Article
- Biomass Fuel Use and Cardiac Function in Nepali Women.Global heart · 2020Article
- C-reactive protein from dried blood spots: Application to household air pollution field studies.Indoor air · 2020Article
- Cooking with biomass fuel and cardiovascular disease: a cross-sectional study among rural villagers in Phitsanulok, Thailand.F1000Research · 2020Article
- Household fuel use and biomarkers of inflammation and respiratory illness among rural South African Women.Environmental research · 2018Article
- Observational
- Systemic serum amyloid A as a biomarker for exposure to zinc and/or copper-containing metal fumes.Journal of exposure science & environmental epidemiology · 2018Article
- Biomass fuel smoke exposure was associated with adverse cardiac remodeling and left ventricular dysfunction in Peru.Indoor air · 2017Article
- C-reactive protein is differentially modulated by co-existing infections, vitamin deficiencies and maternal factors in pregnant and lactating indigenous Panamanian women.Infectious diseases of poverty · 2017Article
- Components of Boiogito Suppress the Progression of Hypercholesterolemia and Fatty Liver Induced by High-Cholesterol Diet in Rats.Yonago acta medica · 2016Article
Corrections and comments
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Authors and funding
10 authors at 3 institutions in 2 countries.
Funding
Abstract
Indoor smoke exposure may affect cardiovascular disease (CVD) risk via lung-mediated inflammation, oxidative stress, and endothelial inflammation. We sought to explore the association between indoor smoke exposure from burning biomass fuels and a selected group of markers for endothelial inflammation. We compared serum concentrations of amyloid A protein, E-selectin, soluble intercellular adhesion molecule 1 (ICAM-1) and VCAM-1, von Willebrand factor (vWF), and high-sensitivity C-reactive protein (hs-CRP) in 228 biomass-exposed vs. 228 non-exposed participants living in Puno, Peru. Average age was 56 years (s.d. = 13), average BMI was 26.5 kg/m(2) (s.d. = 4.4), 48% were male, 59.4% completed high school, and 2% reported a physician diagnosis of CVD. In unadjusted analysis, serum levels of soluble ICAM-1 (330 vs. 302 ng/ml; P < 0.001), soluble VCAM-1 (403 vs. 362 ng/ml; P < 0.001), and E-selectin (54.2 vs. 52.7 ng/ml; P = 0.05) were increased in biomass-exposed vs. non-exposed participants, respectively, whereas serum levels of vWF (1148 vs. 1311 mU/ml; P < 0.001) and hs-CRP (2.56 vs. 3.12 mg/l; P < 0.001) were decreased, respectively. In adjusted analyses, chronic exposure to biomass fuels remained positively associated with serum levels of soluble ICAM-1 (P = 0.03) and VCAM-1 (P = 0.05) and E-selectin (P = 0.05), and remained negatively associated with serum levels of vWF (P = 0.02) and hs-CRP (P < 0.001). Daily exposure to biomass fuel smoke was associated with important differences in specific biomarkers of endothelial inflammation and may help explain accelerated atherosclerosis among those who are chronically exposed.
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Registered trials
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