Evidence map›Paper›PMID 26464246›Full record

ArticleHistochemistry and cell biology2016

Expression of Sulf1 and Sulf2 in cartilage, bone and endochondral fracture healing.

G Zaman, K A Staines, C Farquharson, P T Newton, J Dudhia, C Chenu, A A Pitsillides, G K Dhoot

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In one paragraph

Article in Histochemistry and cell biology, 2016. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 14 papers.

0numbers the graph read from it
0cells of the map it votes in
14citing papers in PubMed
1.6field-weighted citation impact, top 18% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

14 citing papers in PubMed, 31 citations in OpenAlex.

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  11. The histone demethylase JMJD2B regulates endothelial-to-mesenchymal transition.Proceedings of the National Academy of Sciences of the United States of America · 2020
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors at 4 institutions in 2 countries.

G ZamanDepartment of Comparative Biomedical Sciences, RVC, University of London, London, NW1 OTU, UK.
K A StainesThe Roslin Institute, Royal (Dick) School of Veterinary Studies, The University of Edinburgh, Easter Bush, Midlothian, EH25 9RG, UK.
C FarquharsonThe Roslin Institute, Royal (Dick) School of Veterinary Studies, The University of Edinburgh, Easter Bush, Midlothian, EH25 9RG, UK.
P T NewtonThe Roslin Institute, Royal (Dick) School of Veterinary Studies, The University of Edinburgh, Easter Bush, Midlothian, EH25 9RG, UK.
J DudhiaDepartment of Clinical Sciences, RVC, University of London, London, NW1 OTU, UK.
C ChenuDepartment of Comparative Biomedical Sciences, RVC, University of London, London, NW1 OTU, UK.
A A PitsillidesDepartment of Comparative Biomedical Sciences, RVC, University of London, London, NW1 OTU, UK.
G K DhootDepartment of Comparative Biomedical Sciences, RVC, University of London, London, NW1 OTU, UK. tdhoot@rvc.ac.uk.
University of London · GBRoslin Institute · GBUniversidad de Londres · MXUniversity of Edinburgh · GB

Funding

Arthritis Research UK 20581Versus Arthritis 18920
6 · The paper itself

Abstract

SULF1/SULF2 enzymes regulate cell signalling that impacts the growth and differentiation of many tissues. To determine their possible role in cartilage and bone growth or repair, their expression was examined during development and bone fracture healing using RT-PCR and immunochemical analyses. Examination of epiphyseal growth plates revealed differential, inverse patterns of SULF1 and SULF2 expressions, with the former enriched in quiescent and the latter in hypertrophic chondrocyte zones. Markedly higher levels of both SULFs, however, were expressed in osteoblasts actively forming bone when compared with proliferating pre-osteoblasts in the periosteum or the entombed osteocytes which express the lowest levels. The increased expression of Sulf1 and Sulf2 in differentiating osteoblasts was further confirmed by RT-PCR analysis of mRNA levels in rat calvarial osteoblast cultures. SULF1 and SULF2 were expressed in most foetal articular chondrocytes but down-regulated in a larger subset of cells in the post-natal articular cartilage. Unlike adult articular chondrocytes, SULF1/SULF2 expression varied markedly in post-natal hypertrophic chondrocytes in the growth plate, with very high SULF2 expression compared with SULF1 apparent during neonatal growth in both primary and secondary centres of ossification. Similarly, hypertrophic chondrocytes expressed greatly higher levels of SULF2 but not SULF1 during bone fracture healing. SULF2 expression unlike SULF1 also spread to the calcifying matrix around the hypertrophic chondrocytes indicating its possible ligand inhibiting role through HSPG desulphation. Higher levels of SULF2 in both developing and healing bone closely correlated with parallel increases in hedgehog signalling analysed by ptc1 receptor expression.

Indexed as

AnimalsBone and BonesCalcification, PhysiologicCartilage, ArticularCell DifferentiationCells, CulturedChondrocytesChondrogenesisFracture HealingGrowth PlateHumansMaleOsteoblastsOsteocytesOsteogenesisPatched ReceptorsPatched ReceptorsSULF1 protein, humanSULF2 protein, humanSulfatasesSulfotransferasesBoneBone fractureCartilageCell signallingSulf1Sulf2

Identifiers

PMID26464246
OpenAlexW1785098427

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.