Evidence map›Paper›PMID 26431707›Full record

ArticleEuropean radiology2016

Endovascular treatment of complex aortic aneurysms: prevalence of acute kidney injury and effect on long-term renal function.

Anna M Sailer, Patricia J Nelemans, Camille van Berlo, Ozan Yazar, Michiel W de Haan, Dominik Fleischmann, Geert Willem H Schurink

Abstract read
In one paragraph

Article in European radiology, 2016. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 11 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
11citing papers in PubMed, 1 pooled it
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

11 citing papers in PubMed, 1 synthesis or guideline pooled it.

  1. Pooled it
  2. Article
  3. Article
  4. Article
  5. Review
  6. Review
  7. Article
  8. Article
  9. Article
  10. Article
  11. Endovascular Management of Abdominal Aortic Aneurysms: the Year in Review.Current treatment options in cardiovascular medicine · 2016
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors.

Anna M SailerDepartment of Radiology, Maastricht University Medical Centre, Maastricht, The Netherlands. karmanna@stanford.edu.
Patricia J NelemansDepartment of Epidemiology, Maastricht University Medical Centre, Maatsricht, The Netherlands.
Camille van BerloDepartment of Radiology, Maastricht University Medical Centre, Maastricht, The Netherlands.
Ozan YazarDepartment of Vascular Surgery, Cliniques del Europe, Brussels, Belgium.
Michiel W de HaanDepartment of Radiology, Maastricht University Medical Centre, Maastricht, The Netherlands.
Dominik FleischmannDepartment of Radiology, Stanford University Hospitals and Clinics, Stanford, CA, USA.
Geert Willem H SchurinkDepartment of Surgery, Maastricht University Medical Centre, Maastricht, The Netherlands.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

objectivesTo analyse predictors for short- and long-term renal function changes after fenestrated and branched endovascular aortic repair (EVAR).

methodsA total of 157 patients underwent fenestrated and branched EVAR. Procedural intra-arterial iodinated contrast volume was documented. Serum creatinine and estimated glomerular filtration rate (eGFR) at baseline, during 48 h following EVAR, at discharge and latest moment of follow-up were recorded. Development of post-EVAR acute kidney injury (AKI; according to AKIN criteria), and potential risk factors for renal failure were recorded. Multivariate regression analyses were used to identify independent risk factors for AKI and eGFR decrease during follow-up.

resultsForty-three patients (28 %) developed post-EVAR AKI. Long procedure time and occlusion of accessory renal arteries were independent risk factors for development of AKI. (odds ratio (OR) 1.005 per minute, 95 % CI 1.001-1.01; p = 0.025 and OR 3.02, 95 % CI 1.19-8.16; p = 0.029). Post-EVAR AKI was associated with a significantly increased risk for eGFR decrease at discharge and latest follow-up (hazard ratio (HR) 3.47, 95 % CI 1.63-7.36, p = 0.001 and HR 3.01, 95 % CI 1.56-5.80; p = 0.001). Iodinated contrast volume was not an independent risk factor for AKI or eGFR decrease during follow-up.

conclusionDevelopment of post-EVAR AKI is an independent risk factor for long-term renal function decrease. KEY POINTS: • Longer procedure time is associated with an increased risk for AKI. • Renal perfusion defects on angiography are associated with increased risk for AKI. • Post-EVAR AKI is associated with higher probability for long-term eGFR decrease. • Iodinated contrast volume is not an independent risk factor for AKI. • Iodinated contrast volume is not an independent risk factor for long-term eGFR decrease.

Indexed as

Acute Kidney InjuryAgedAortic AneurysmCreatinineEndovascular ProceduresFemaleFollow-Up StudiesGlomerular Filtration RateHumansKidneyKidney Function TestsMalePrevalenceRetrospective StudiesRisk FactorsTimeCreatinineAcute kidney injuryAortic aneurysmContrast mediaEndovascular proceduresGlomerular filtration rate

Identifiers

PMID26431707
PMCPMC4863901

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.