ArticleVirulence2015
Understanding Mycobacterium avium subspecies hominissuis microaggregate mediated pathogenesis.
Article in Virulence, 2015. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
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1 author.
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Abstract
Mycobacterium avium subspecies hominissuis (MAH) is an opportunistic pathogen and causes nontuberculous infections in immune compromised individuals, an emerging problem that has been recognized worldwide. Understanding the pathogenesis of this organism is important as better treatment and prevention options are needed. Microaggregates form when two or more bacterial cells join at a surface. MAH forms micgroaggregates to promote its entry in to epithelial cells and cause infection. The mechanisms involved in the interaction between the microaggregate and the host are becoming clearer as the molecules involved in this process are being uncovered. Microaggregate Invasion Protein-1 (MIP-1) is now described as having a major role in the invasion of epithelial cells by MAH.
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.