Evidence map›Paper›PMID 26354912›Full record

ArticleThe Journal of neuroscience : the official journal of the Society for Neuroscience2015

Neuronal Heterotopias Affect the Activities of Distant Brain Areas and Lead to Behavioral Deficits.

Kazuhiro Ishii, Ken-ichiro Kubo, Toshihiro Endo, Keitaro Yoshida, Seico Benner, Yukiko Ito, Hidenori Aizawa, Michihiko Aramaki, Akihiro Yamanaka, Kohichi Tanaka and 6 more

Open access · bronzeAbstract read
In one paragraph

Article in The Journal of neuroscience : the official journal of the Society for Neuroscience, 2015. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 23 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
23citing papers in PubMed, 1 pooled it
2.5field-weighted citation impact, top 11% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

23 citing papers in PubMed, 1 synthesis or guideline pooled it, 43 citations in OpenAlex.

  1. Pooled it
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  5. Review
  6. Article
  7. Article
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  16. Neural Correlates of Sensory Abnormalities Across Developmental Disabilities.International review of research in developmental disabilities · 2018
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  18. Reelin transiently promotes N-cadherin-dependent neuronal adhesion during mouse cortical development.Proceedings of the National Academy of Sciences of the United States of America · 2017
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  20. Resolving Behavioral Output via Chemogenetic Designer Receptors Exclusively Activated by Designer Drugs.The Journal of neuroscience : the official journal of the Society for Neuroscience · 2016
    Review
4 · The record

Corrections and comments

5 · Who and what money

Authors and funding

16 authors at 7 institutions in 3 countries.

Kazuhiro IshiiDepartment of Anatomy, Keio University School of Medicine, Tokyo, 160-8582, Japan.ORCID http://orcid.org/0000-0002-3234-1446
Ken-ichiro KuboDepartment of Anatomy, Keio University School of Medicine, Tokyo, 160-8582, Japan.ORCID http://orcid.org/0000-0003-4023-5069
Toshihiro EndoLaboratory of Environmental Health Sciences, Center for Disease Biology and Integrative Medicine, Graduate School of Medicine, and Department of Neurochemistry, Graduate School of Medicine, University of Tokyo, Tokyo, 113-0033, Japan.
Keitaro YoshidaDepartment of Neuropsychiatry, Keio University School of Medicine, Tokyo, 160-8582, Japan.
Seico BennerLaboratory of Environmental Health Sciences, Center for Disease Biology and Integrative Medicine, Graduate School of Medicine, and.
Yukiko ItoLaboratory of Molecular Neuroscience, Medical Research Institute, Tokyo Medical and Dental University, Tokyo, 113-8510, Japan.
Hidenori AizawaLaboratory of Molecular Neuroscience, Medical Research Institute, Tokyo Medical and Dental University, Tokyo, 113-8510, Japan, Department of Neurobiology, Institute of Biomedical and Health Sciences, Hiroshima University, Hiroshima, 734-8553, Japan.ORCID http://orcid.org/0000-0002-2896-1298
Michihiko AramakiDepartment of Anatomy, Keio University School of Medicine, Tokyo, 160-8582, Japan.
Akihiro YamanakaDepartment of Neuroscience II, Research Institute of Environmental Medicine, Nagoya University, Nagoya, 464-8601, Japan.ORCID http://orcid.org/0000-0001-6099-7306
Kohichi TanakaLaboratory of Molecular Neuroscience, Medical Research Institute, Tokyo Medical and Dental University, Tokyo, 113-8510, Japan, Center for Brain Integration Research, Tokyo Medical and Dental University, Tokyo, 113-8510, Japan, and.
Norio TakataDepartment of Neuropsychiatry, Keio University School of Medicine, Tokyo, 160-8582, Japan.
Kenji F TanakaDepartment of Neuropsychiatry, Keio University School of Medicine, Tokyo, 160-8582, Japan.
Masaru MimuraDepartment of Neuropsychiatry, Keio University School of Medicine, Tokyo, 160-8582, Japan.
Chiharu TohyamaLaboratory of Environmental Health Sciences, Center for Disease Biology and Integrative Medicine, Graduate School of Medicine, and.
Masaki KakeyamaLaboratory of Environmental Health Sciences, Center for Disease Biology and Integrative Medicine, Graduate School of Medicine, and Laboratory for Systems Neuroscience and Preventive Medicine, Waseda University Faculty of Human Sciences, Saitama, 359-1192, Japan kazunori@keio.jp kake@waseda.jp.ORCID http://orcid.org/0000-0001-9063-9673
Kazunori NakajimaDepartment of Anatomy, Keio University School of Medicine, Tokyo, 160-8582, Japan, kazunori@keio.jp kake@waseda.jp.
Keio University · JPLaboratoire d’immunologie intégrative du cancer · FRTokyo Medical and Dental University · JPHiroshima University · JPNagoya University · JPThe University of Tokyo · JPWaseda University · JP

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Neuronal heterotopia refers to brain malformations resulting from deficits of neuronal migration. Individuals with heterotopias show a high incidence of neurological deficits, such as epilepsy. More recently, it has come to be recognized that focal heterotopias may also show a range of psychiatric problems, including cognitive and behavioral impairments. However, because focal heterotopias are not always located in the brain areas responsible for the symptoms, the causal relationship between the symptoms and heterotopias remains elusive. In this study, we showed that mice with focal heterotopias in the somatosensory cortex generated by in utero electroporation exhibited spatial working memory deficit and low competitive dominance behavior, which have been shown to be closely associated with the activity of the medial prefrontal cortex (mPFC) in rodents. Analysis of the mPFC activity revealed that the immediate-early gene expression was decreased and the local field potentials of the mPFC were altered in the mice with heterotopias compared with the control mice. Moreover, activation of these ectopic and overlying sister neurons using the DREADD (designer receptor exclusively activated by designer drug) system improved the working memory deficits. These findings suggest that cortical regions containing focal heterotopias can affect distant brain regions and give rise to behavioral abnormalities. Significance statement: Recent studies reported that patients with heterotopias have a variety of clinical symptoms, such as cognitive disturbance, psychiatric symptoms, and autistic behavior. However, the causal relationship between the symptoms and heterotopias remains elusive. Here we showed that mice with focal heterotopias in the somatosensory cortex generated by in utero electroporation exhibited behavioral deficits that have been shown to be associated with the mPFC activity in rodents. The existence of heterotopias indeed altered the neural activities of the mPFC, and direct manipulation of the neural activity of the ectopic neurons and their sister neurons in the overlying cortex improved the behavioral deficit. Thus, our results indicate that focal heterotopias could affect the activities of distant brain areas and cause behavioral abnormalities.

Indexed as

AnimalsGenes, Immediate-EarlyMalformations of Cortical DevelopmentMaze LearningMemoryMental DisordersMicePrefrontal CortexSocial BehaviorSomatosensory CortexDREADDheterotopiaIntelliCagein utero electroporationneuropsychiatric symptomoptogenetics

Identifiers

PMID26354912
PMCPMC6605399
OpenAlexW2159778294

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.