ArticleJournal of exposure science & environmental epidemiology
Ambient air pollution and emergency department visits for asthma: a multi-city assessment of effect modification by age.
Article in Journal of exposure science & environmental epidemiology. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 41 papers, 3 of them syntheses that pooled it.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
41 citing papers in PubMed, 3 syntheses or guidelines pooled it.
- The Impact of Ambient Ozone Exposure on Respiratory Health in the U.S. Populations: A Systematic Review.Current environmental health reports · 2026Pooled it
- Update of a systematic review on associations between environmental exposures and asthma control and exacerbations in young children.BMJ open · 2025Pooled it
- Short-term Effect of Fine Particulate Matter on Children's Hospital Admissions and Emergency Department Visits for Asthma: A Systematic Review and Meta-analysis.Journal of preventive medicine and public health = Yebang Uihakhoe chi · 2016Pooled it
- A Policy Support Analysis of Air Pollution Epidemiology and Hospital Admissions in Kuwait.Journal of epidemiology and global health · 2026Article
- Article
- Modeling Joint Health Effects of Environmental Exposure Mixtures With Bayesian Additive Regression Trees.Statistics in medicine · 2025Article
- Inhalation of concentrated ambient PM2.5 promotes inactivation of telomerase reverse transcriptase, telomere shortening, and senescence of multiple cell types in mice.Toxicological sciences : an official journal of the Society of Toxicology · 2025Article
- Sex and age characteristics of thunderstorm asthma emergency department visits.Hygiene and environmental health advances · 2024Article
- The Role of Neighborhood Air Pollution in Disparate Racial and Ethnic Asthma Acute Care Use.American journal of respiratory and critical care medicine · 2024Article
- Air Quality and Health Impacts of Onshore Oil and Gas Flaring and Venting Activities Estimated Using Refined Satellite-Based Emissions.GeoHealth · 2024Article
- A scalar-on-quantile-function approach for estimating short-term health effects of environmental exposures.Biometrics · 2024Article
- Primary Care Asthma Attack Prediction Models for Adults: A Systematic Review of Reported Methodologies and Outcomes.Journal of asthma and allergy · 2024Review
- Impacts of residential indoor air quality and environmental risk factors on adult asthma-related health outcomes in Chicago, IL.Journal of exposure science & environmental epidemiology · 2023Article
- Acute Effects of Ambient Air Pollution on Asthma Emergency Department Visits in Ten U.S. States.Environmental health perspectives · 2023Article
- Urinary metabolites of polycyclic aromatic hydrocarbons and short-acting beta agonist or systemic corticosteroid asthma medication use within NHANES.Environmental research · 2023Article
- Effect of PM2.5 Levels on ED Visits for Respiratory Causes in a Greek Semi-Urban Area.Journal of personalized medicine · 2022Article
- Unencapsulated and washable two-dimensional material electronic-textile for NOScientific reports · 2022Article
- The association between asthma emergency department visits and satellite-derived PMEnvironmental research · 2021Article
- Article
- How Do Inflammatory Mediators, Immune Response and Air Pollution Contribute to COVID-19 Disease Severity? A Lesson to Learn.Life (Basel, Switzerland) · 2021Review
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
6 authors.
Funding
Abstract
Previous studies have found strong associations between asthma morbidity and major ambient air pollutants. Relatively little research has been conducted to assess whether age is a factor conferring susceptibility to air pollution-related asthma morbidity. We investigated the short-term relationships between asthma emergency department (ED) visits and ambient ozone (O3), carbon monoxide (CO), nitrogen dioxide (NO2), sulfur dioxide (SO2), and fine particulate matter (PM2.5) in Atlanta (1993-2009), Dallas (2006-2009), and St. Louis (2001-2007). City-specific daily time-series analyses were conducted to estimate associations by age group (0-4, 5-18, 19-39, 40-64, and 65+ years). Sub-analyses were performed stratified by race and sex. City-specific rate ratios (RRs) were combined by inverse-variance weighting to provide an overall association for each strata. The overall RRs differed across age groups, with associations for all pollutants consistently strongest for children aged 5-18 years. The patterns of association across age groups remained generally consistent when models were stratified by sex and race, although the strong observed associations among 5-18 year olds appeared to be partially driven by non-white and male patients. Our findings suggest that age is a susceptibility factor for asthma exacerbations in response to air pollution, with school-age children having the highest susceptibility.
Indexed as
Identifiers
26350981What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.