Evidence map›Paper›PMID 26335371›Full record

ArticleThe international journal of cardiovascular imaging2016

Relationship between epicardial fat and quantitative coronary artery plaque progression: insights from computer tomography coronary angiography.

Peter J Psaltis, Andrew H Talman, Kiran Munnur, James D Cameron, Brian S H Ko, Ian T Meredith, Sujith K Seneviratne, Dennis T L Wong

Abstract read
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In one paragraph

Article in The international journal of cardiovascular imaging, 2016. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 11 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
11citing papers in PubMed, 1 pooled it
1.5field-weighted citation impact, top 17% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

11 citing papers in PubMed, 1 synthesis or guideline pooled it, 23 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors at 2 institutions in 1 country.

Peter J PsaltisMonash Heart, Monash Cardiovascular Research Centre, Monash University, Clayton, VIC, Australia.
Andrew H TalmanMonash Heart, Monash Cardiovascular Research Centre, Monash University, Clayton, VIC, Australia.
Kiran MunnurMonash Heart, Monash Cardiovascular Research Centre, Monash University, Clayton, VIC, Australia.
James D CameronMonash Heart, Monash Cardiovascular Research Centre, Monash University, Clayton, VIC, Australia.
Brian S H KoMonash Heart, Monash Cardiovascular Research Centre, Monash University, Clayton, VIC, Australia.
Ian T MeredithMonash Heart, Monash Cardiovascular Research Centre, Monash University, Clayton, VIC, Australia.
Sujith K SeneviratneMonash Heart, Monash Cardiovascular Research Centre, Monash University, Clayton, VIC, Australia.
Dennis T L WongMonash Heart, Monash Cardiovascular Research Centre, Monash University, Clayton, VIC, Australia. drdenniswong@yahoo.com.au.
Monash University · AUSouth Australian Health and Medical Research Institute · AU

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Epicardial fat volume (EFV) has been suggested to promote atherosclerotic plaque development in coronary arteries, and has been correlated with both coronary stenosis and acute coronary events. Although associated with progression of coronary calcification burden, a relationship with progression of coronary atheroma volume has not been previously tested. We studied patients who had clinically indicated serial 320-row multi-detector computer tomography coronary angiography with a median 25-month interval. EFV was measured at baseline and follow-up. In vessels with coronary stenosis, quantitative analysis was performed to measure atherosclerotic plaque burden, volume and aggregate plaque volume at baseline and follow-up. The study comprised 64 patients (58.4 ± 12.2 years, 27 males, 192 vessels, 193 coronary segments). 79 (41 %) coronary segments had stenosis at baseline. Stenotic segments were associated with greater baseline EFV than those without coronary stenosis (117.4 ± 45.1 vs. 102.3 ± 51.6 cm(3), P = 0.046). 46 (24 %) coronary segments displayed either new plaque formation or progression of adjusted plaque burden at follow-up. These were associated with higher baseline EFV than segments without stenosis or those segments that had stenoses that did not progress (128.7 vs. 101.0 vs. 106.7 cm(3) respectively, P = 0.006). On multivariate analysis, baseline EFV was the only independent predictor of coronary atherosclerotic plaque progression or new development (P = 0.014). High baseline EFV is associated with the presence of coronary artery stenosis and plaque volume progression. Accumulation of EFV may be implicated in the evolution and progression of coronary atheroma.

Indexed as

Coronary AngiographyMultidetector Computed TomographyAdipose TissueCoronary Artery DiseaseCoronary StenosisDisease ProgressionFemaleHumansMaleMiddle AgedPlaque, AtheroscleroticRetrospective StudiesRisk FactorsCardiac CTComputed tomographyCoronary artery plaque progressionCoronary artery stenosisEpicardial fat

Identifiers

PMID26335371
OpenAlexW1262413770

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.