Evidence map›Paper›PMID 26284086›Full record

ArticleFrontiers in plant science2015

Functional analysis of the BRI1 receptor kinase by Thr-for-Ser substitution in a regulatory autophosphorylation site.

Man-Ho Oh, Kyle W Bender, Sang Y Kim, Xia Wu, Seulki Lee, Ill-Sup Nou, Raymond E Zielinski, Steven D Clouse, Steven C Huber

Open access · goldAbstract read
In one paragraph

Article in Frontiers in plant science, 2015. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers.

0numbers the graph read from it
0cells of the map it votes in
5citing papers in PubMed
2.1field-weighted citation impact, top 12% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

5 citing papers in PubMed, 11 citations in OpenAlex.

  1. Activation loop phosphorylation of a non-RD receptor kinase initiates plant innate immune signaling.Proceedings of the National Academy of Sciences of the United States of America · 2021
    Article
  2. In vivo evidence for a regulatory role of phosphorylation ofProceedings of the National Academy of Sciences of the United States of America · 2019
    Article
  3. SlBIR3 Negatively Regulates PAMP Responses and Cell Death in Tomato.International journal of molecular sciences · 2017
    Article
  4. Article
  5. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors at 1 institution in 1 country.

Man-Ho OhPlant Developmental Genetics, Department of Biological Science, College of Biological Sciences and Biotechnology, Chungnam National University Daejeon, South Korea ; Protein Biochemistry, Department of Plant Biology, University of Illinois Urbana, IL, USA.
Kyle W BenderProtein Biochemistry, Department of Plant Biology, University of Illinois Urbana, IL, USA.
Sang Y KimProtein Biochemistry, Department of Plant Biology, University of Illinois Urbana, IL, USA ; U.S. Department of Agriculture, Agricultural Research Service Urbana, IL, USA.
Xia WuDepartment of Genome Sciences, University of Washington Seattle, WA, USA.
Seulki LeePlant Developmental Genetics, Department of Biological Science, College of Biological Sciences and Biotechnology, Chungnam National University Daejeon, South Korea.
Ill-Sup NouDepartment of Horticulture, Sunchon National University Sunchon, South Korea.
Raymond E ZielinskiProtein Biochemistry, Department of Plant Biology, University of Illinois Urbana, IL, USA.
Steven D ClouseDepartment of Horticultural Science, NC State University Raleigh, NC, USA.
Steven C HuberProtein Biochemistry, Department of Plant Biology, University of Illinois Urbana, IL, USA ; U.S. Department of Agriculture, Agricultural Research Service Urbana, IL, USA.
University of Washington · US

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

BRI1 becomes highly phosphorylated in vivo upon perception of the ligand, brassinolide, as a result of autophosphorylation and transphosphorylation by its co-receptor kinase, BAK1. Important autophosphorylation sites include those involved in activation of kinase activity and those that are inhibitory, such as Ser-891. The inhibitory sites are autophosphorylated after kinase activation has been achieved and are postulated to contribute to deactivation of the kinase. The function of phosphosites is usually tested by substituting a non-phosphorylatable residue or an acidic residue that can act as a phosphomimetic. What has typically not been examined is substitution of a Thr for a Ser phosphosite (or vice versa) but given that Thr and Ser are not equivalent amino acids this type of substitution may represent a new approach to engineer regulatory phosphorylation. In the present study with BRI1, we substituted Thr at the Ser-891 phosphosite to generate the S891T directed mutant. The recombinant Flag-BRI1 (S891T) cytoplasmic domain protein (the S891T protein) was catalytically active and phosphorylation occurred at the engineered Thr-891 site. However, the S891T recombinant protein autophosphorylated more slowly than the wild-type protein during expression in E. coli. As a result, activation of peptide kinase activity (measured in vitro) was delayed as was transphosphorylation of bacterial proteins in situ. Stable transgenic expression of BRI1 (S891T)-Flag in Arabidopsis bri1-5 plants did not fully rescue the brassinosteroid (BR) phenotype indicating that BR signaling was constrained. Our working model is that restricted signaling in the S891T plants occurs as a result of the reduced rate of activation of the mutant BRI1 kinase by autophosphorylation. These results provide the platform for future studies to critically test this new model in vivo and establish Ser-Thr substitutions at phosphosites as an interesting approach to consider with other protein kinases.

Indexed as

autophosphorylationBRI1directed mutagenesiskinase domain

Identifiers

PMID26284086
PMCPMC4519688
OpenAlexW1554179876

What OpenQuestion holds

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LicenceCC BY
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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.